Hypermineralocorticoid: Difference between revisions
No edit summary |
Kiran Singh (talk | contribs) No edit summary |
||
| (2 intermediate revisions by 2 users not shown) | |||
| Line 1: | Line 1: | ||
{{Infobox_Disease | | |||
Name = {{PAGENAME}} | | |||
Image = | | |||
Caption = | | |||
DiseasesDB = | | |||
ICD10 = | | |||
ICD9 = | | |||
ICDO = | | |||
OMIM = | | |||
MedlinePlus = | | |||
eMedicineSubj = | | |||
eMedicineTopic = | | |||
MeshID = | | |||
}} | |||
{{SI}} | |||
{{CMG}} | |||
==Overview== | |||
'''Hypermineralocorticoid''' is a hereditary disease characterized by childhood onset hypertension, hypokalemic alkalosis, and low renin and aldosterone secretion. It results from a defect in the activity of the 11-beta-hydroxysteroid dehydrogenase type 2 enzyme which results in inadequate conversion of cortisol to cortisone. The build up of unprocessed cortisol to levels that stimulate mineralocorticoid receptors creates the appearance of having excessive mineralocorticoids. | '''Hypermineralocorticoid''' is a hereditary disease characterized by childhood onset hypertension, hypokalemic alkalosis, and low renin and aldosterone secretion. It results from a defect in the activity of the 11-beta-hydroxysteroid dehydrogenase type 2 enzyme which results in inadequate conversion of cortisol to cortisone. The build up of unprocessed cortisol to levels that stimulate mineralocorticoid receptors creates the appearance of having excessive mineralocorticoids. | ||
{{Symptoms and signs}} | {{Symptoms and signs}} | ||
{{WikiDoc Help Menu}} | {{WikiDoc Help Menu}} | ||
{{WikiDoc Sources}} | {{WikiDoc Sources}} | ||
Latest revision as of 18:22, 8 June 2015
| Hypermineralocorticoid |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Hypermineralocorticoid is a hereditary disease characterized by childhood onset hypertension, hypokalemic alkalosis, and low renin and aldosterone secretion. It results from a defect in the activity of the 11-beta-hydroxysteroid dehydrogenase type 2 enzyme which results in inadequate conversion of cortisol to cortisone. The build up of unprocessed cortisol to levels that stimulate mineralocorticoid receptors creates the appearance of having excessive mineralocorticoids.
Template:Skin and subcutaneous tissue symptoms and signs Template:Nervous and musculoskeletal system symptoms and signs Template:Urinary system symptoms and signs Template:Cognition, perception, emotional state and behaviour symptoms and signs Template:Speech and voice symptoms and signs Template:General symptoms and signs