Pneumatosis intestinalis pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
There are two theories on pathogenesis of pneumatois intestinalis. Mechanical theory suggests that gas dissects into the bowel wall from either the intestinal lumen or the lungs via the mediastinum due to some mechanism causing increased pressure. Bacterial theory proposes that gas-forming bacilli enter the submucosa through mucosal rents or increased mucosal permeability and produce gas within the bowel wall. | There are two theories on pathogenesis of pneumatois intestinalis. Mechanical theory suggests that gas dissects into the [[bowel]] wall from either the intestinal lumen or the [[lungs]] via the [[mediastinum]] due to some mechanism causing increased pressure. Bacterial theory proposes that gas-forming [[bacilli]] enter the submucosa through mucosal rents or increased mucosal permeability and produce gas within the bowel wall. | ||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
[[Category:Gastroenterology]] | [[Category:Gastroenterology]] | ||
[[Category:Radiologic signs]] | [[Category:Radiologic signs]] | ||
[[Category:Radiology]] | [[Category:Radiology]] | ||
[[Category:Needs content]] | [[Category:Needs content]] | ||
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Latest revision as of 14:45, 12 June 2015
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Pneumatosis Intestinalis Microchapters |
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Differentiating Pneumatosis Intestinalis from other Conditions |
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Diagnosis |
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Treatment |
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Pneumatosis intestinalis pathophysiology On the Web |
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Overview
There are two theories on pathogenesis of pneumatois intestinalis. Mechanical theory suggests that gas dissects into the bowel wall from either the intestinal lumen or the lungs via the mediastinum due to some mechanism causing increased pressure. Bacterial theory proposes that gas-forming bacilli enter the submucosa through mucosal rents or increased mucosal permeability and produce gas within the bowel wall.