Porphyria cutanea tarda classification: Difference between revisions
Jump to navigation
Jump to search
(Created page with "__NOTOC__ ==Overview== '''Porphyria cutanea tarda''' (PCT) is the most common subtype of porphyria. The disorder results from low levels of the enzyme responsible for...") |
Irfan Dotani (talk | contribs) No edit summary |
||
Line 1: | Line 1: | ||
__NOTOC__ | __NOTOC__ | ||
{{Porphyria cutanea tarda}} | |||
{{CMG}}; {{AE}} | |||
==Overview== | ==Overview== | ||
'''Porphyria cutanea tarda''' (PCT) is the most common subtype of [[porphyria]]. The disorder results from low levels of the [[enzyme]] responsible for the [[uroporphyrinogen III decarboxylase|fifth step]] in [[heme]] production. Heme is a vital molecule for all of the body's organs. It is a component of [[hemoglobin]], the molecule that carries [[oxygen]] in the blood. | '''Porphyria cutanea tarda''' (PCT) is the most common subtype of [[porphyria]]. The disorder results from low levels of the [[enzyme]] responsible for the [[uroporphyrinogen III decarboxylase|fifth step]] in [[heme]] production. Heme is a vital molecule for all of the body's organs. It is a component of [[hemoglobin]], the molecule that carries [[oxygen]] in the blood. | ||
== | ==Classification== | ||
*In the acquired form, the signs and symptoms of this condition are triggered by non genetic factors such as alcohol abuse, excess iron, certain hormones, and viral infections. However, in the inherited form of the disease, which affects up to 20% of PCT patients, the condition arises from a mutation in the uroporphyrinogen decarboxylase gene, although environmental and chemical factors may trigger or exacerbate symptoms. | *In the acquired form, the signs and symptoms of this condition are triggered by non genetic factors such as alcohol abuse, excess iron, certain hormones, and viral infections. However, in the inherited form of the disease, which affects up to 20% of PCT patients, the condition arises from a mutation in the uroporphyrinogen decarboxylase gene, although environmental and chemical factors may trigger or exacerbate symptoms. | ||
*Drug side effect: [[Chlorpropamide]], [[Tolbutamide]], [[Tolazamide]] | *Drug side effect: [[Chlorpropamide]], [[Tolbutamide]], [[Tolazamide]] | ||
Line 9: | Line 12: | ||
==References== | ==References== | ||
{{reflist}} | {{reflist}} | ||
[[Category:Endocrinology]] | |||
{{WS}} | |||
{{WH}} |
Latest revision as of 19:23, 25 July 2016
Porphyria cutanea tarda Microchapters |
Diagnosis |
---|
Treatment |
Case Studies |
Porphyria cutanea tarda classification On the Web |
American Roentgen Ray Society Images of Porphyria cutanea tarda classification |
Risk calculators and risk factors for Porphyria cutanea tarda classification |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
Overview
Porphyria cutanea tarda (PCT) is the most common subtype of porphyria. The disorder results from low levels of the enzyme responsible for the fifth step in heme production. Heme is a vital molecule for all of the body's organs. It is a component of hemoglobin, the molecule that carries oxygen in the blood.
Classification
- In the acquired form, the signs and symptoms of this condition are triggered by non genetic factors such as alcohol abuse, excess iron, certain hormones, and viral infections. However, in the inherited form of the disease, which affects up to 20% of PCT patients, the condition arises from a mutation in the uroporphyrinogen decarboxylase gene, although environmental and chemical factors may trigger or exacerbate symptoms.
- Drug side effect: Chlorpropamide, Tolbutamide, Tolazamide