Staphylococcal scalded skin syndrome pathophysiology: Difference between revisions
Created page with "__NOTOC__ {{SSSS}} Please help WikiDoc by adding content here. It's easy! Click here to learn about editing. ==References== {{Reflist|2}} [[..." |
m Changes made per Mahshid's request |
||
(One intermediate revision by one other user not shown) | |||
Line 1: | Line 1: | ||
__NOTOC__ | __NOTOC__ | ||
{{SSSS}} | {{SSSS}} | ||
{{CMG}} | |||
Please help WikiDoc by adding content here. It's easy! Click [[Help:How_to_Edit_a_Page|here]] to learn about editing. | Please help WikiDoc by adding more content here. It's easy! Click [[Help:How_to_Edit_a_Page|here]] to learn about editing. | ||
==Overview== | |||
The [[syndrome]] is induced by epidermolytic [[exotoxin]]s ([[exfoliatin]])<ref>{{DorlandsDict|three/000037652|exfoliatin}}</ref> A and B, which are released by ''S. aureus'' and cause detachment within the epidermal layer; by breaking down tight-junctions. One of the exotoxins is produced by the bacterial chromosome, while the other is produced by a [[plasmid]]. (Bacterial plasmids are pieces of self-replicating DNA that often code for secondary characteristics, such as antibiotic resistance, and toxin production.) These exotoxins are proteases that cleave desmoglein-1, which normally holds the [[Stratum granulosum|granulosum]] and [[Stratum spinosum|spinosum]] layers together. | |||
==References== | ==References== | ||
Line 10: | Line 13: | ||
[[Category:Bacterial diseases]] | [[Category:Bacterial diseases]] | ||
[[Category:Bacterium-related cutaneous conditions]] | [[Category:Bacterium-related cutaneous conditions]] | ||
{{WH}} | {{WH}} | ||
{{WS}} | {{WS}} |
Latest revision as of 18:51, 18 September 2017
Staphylococcal scalded skin syndrome Microchapters |
Differentiating Staphylococcal scalded skin syndrome from other Diseases |
---|
Diagnosis |
Treatment |
Case Studies |
Staphylococcal scalded skin syndrome pathophysiology On the Web |
American Roentgen Ray Society Images of Staphylococcal scalded skin syndrome pathophysiology |
Staphylococcal scalded skin syndrome pathophysiology in the news |
Blogs on Staphylococcal scalded skin syndrome pathophysiology |
Directions to Hospitals Treating Staphylococcal scalded skin syndrome |
Risk calculators and risk factors for Staphylococcal scalded skin syndrome pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Please help WikiDoc by adding more content here. It's easy! Click here to learn about editing.
Overview
The syndrome is induced by epidermolytic exotoxins (exfoliatin)[1] A and B, which are released by S. aureus and cause detachment within the epidermal layer; by breaking down tight-junctions. One of the exotoxins is produced by the bacterial chromosome, while the other is produced by a plasmid. (Bacterial plasmids are pieces of self-replicating DNA that often code for secondary characteristics, such as antibiotic resistance, and toxin production.) These exotoxins are proteases that cleave desmoglein-1, which normally holds the granulosum and spinosum layers together.
References