Toxic multinodular goiter: Difference between revisions
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==[[Toxic multinodular goiter pathophysiology|Pathophysiology]]== | ==[[Toxic multinodular goiter pathophysiology|Pathophysiology]]== | ||
The progression to Toxic multinodular goiter usually involves the somatic gain-of-function mutations in the TSH receptor in autonomously functioning thyroid nodules. | |||
==[[Toxic multinodular goiter causes|Causes]]== | ==[[Toxic multinodular goiter causes|Causes]]== | ||
Latest revision as of 16:28, 6 December 2017
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Toxic multinodular goiter Microchapters |
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Differentiating Toxic multinodular goiter from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Toxic multinodular goiter On the Web |
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American Roentgen Ray Society Images of Toxic multinodular goiter |
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Risk calculators and risk factors for Toxic multinodular goiter |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Ajay Gade MD[2]] Aravind Reddy Kothagadi M.B.B.S[3] Mazia Fatima, MBBS [4]Sunny Kumar MD [5]
Synonyms and keywords:
Overview
Historical Perspective
Classification
Pathophysiology
The progression to Toxic multinodular goiter usually involves the somatic gain-of-function mutations in the TSH receptor in autonomously functioning thyroid nodules.
Causes
Differentiating Toxic multinodular goiter from other Diseases
Epidemiology and Demographics
Risk Factors
Screening
Natural History, Complications and Prognosis
Diagnosis
History and Symptoms | Physical Examination | Laboratory Findings | Electrocardiogram | X-Ray Findings | CT-Scan Findings | MRI Findings | Other Diagnostic Studies | Other Imaging Findings
Treatment
Medical Therapy | Surgery | | Radiation Therapy |Primary Prevention | Secondary Prevention | Cost-Effectiveness of Therapy | Future or Investigational Therapies
Case Studies