IgA nephropathy physical examination: Difference between revisions

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{{IgA nephropathy}}
{{IgA nephropathy}}
{{CMG}}; {{AE}}
{{CMG}}; {{AE}} {{SH}}


==Overview==
==Overview==
Patients with [disease name] usually appear [general appearance]. Physical examination of patients with [disease name] is usually remarkable for [finding 1], [finding 2], and [finding 3].
Patients with IgA nephropathy usually appear normal and usually have no significant clinical finding upon physical examination. However, some of the patients may present with low-grade [[fever]], [[Hypertension|high blood pressure]] with normal [[pulse pressure]], and pitting [[edema]] of the lower extremities in the late stage if the patient develops [[ESRD]].
 
OR
 
Common physical examination findings of [disease name] include [finding 1], [finding 2], and [finding 3].
 
OR
 
The presence of [finding(s)] on physical examination is diagnostic of [disease name].
 
OR
 
The presence of [finding(s)] on physical examination is highly suggestive of [disease name].


==Physical Examination==
==Physical Examination==
* Physical examination of patients with IgA nephropathy is usually normal.
* Physical examination of patients with IgA nephropathy is usually normal.<ref name="pmid7723227">{{cite journal |vauthors=Galla JH |title=IgA nephropathy |journal=Kidney Int. |volume=47 |issue=2 |pages=377–87 |date=February 1995 |pmid=7723227 |doi= |url=}}</ref><ref name="pmid12213946">{{cite journal |vauthors=Donadio JV, Grande JP |title=IgA nephropathy |journal=N. Engl. J. Med. |volume=347 |issue=10 |pages=738–48 |date=September 2002 |pmid=12213946 |doi=10.1056/NEJMra020109 |url=}}</ref><ref name="pmid15524056">{{cite journal |vauthors=Hall CL, Bradley R, Kerr A, Attoti R, Peat D |title=Clinical value of renal biopsy in patients with asymptomatic microscopic hematuria with and without low-grade proteinuria |journal=Clin. Nephrol. |volume=62 |issue=4 |pages=267–72 |date=October 2004 |pmid=15524056 |doi= |url=}}</ref><ref name="pmid8041865">{{cite journal |vauthors=Topham PS, Harper SJ, Furness PN, Harris KP, Walls J, Feehally J |title=Glomerular disease as a cause of isolated microscopic haematuria |journal=Q. J. Med. |volume=87 |issue=6 |pages=329–35 |date=June 1994 |pmid=8041865 |doi= |url=}}</ref><ref name="GutierrezGonzalez2006">{{cite journal|last1=Gutierrez|first1=E.|last2=Gonzalez|first2=E.|last3=Hernandez|first3=E.|last4=Morales|first4=E.|last5=Martinez|first5=M. A.|last6=Usera|first6=G.|last7=Praga|first7=M.|title=Factors That Determine an Incomplete Recovery of Renal Function in Macrohematuria-Induced Acute Renal Failure of IgA Nephropathy|journal=Clinical Journal of the American Society of Nephrology|volume=2|issue=1|year=2006|pages=51–57|issn=1555-9041|doi=10.2215/CJN.02670706}}</ref>


===Appearance of the Patient===
===Appearance of the Patient===
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===Vital Signs===
===Vital Signs===
*Low-grade fever
*Low-grade [[fever]]
*High blood pressure with normal pulse pressure
*[[Hypertension|High blood pressure]] with normal [[pulse pressure]]


===Skin===
===Skin===
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===Heart===
===Heart===
* Cardiovascular examination of patients with [disease name] is usually normal.
* Cardiovascular examination of patients with IgA nephropathy is usually normal.
OR
*Chest tenderness upon palpation
*PMI within 2 cm of the sternum  (PMI) / Displaced point of maximal impulse (PMI) suggestive of ____
*[[Heave]] / [[thrill]]
*[[Friction rub]]
*[[Heart sounds#First heart tone S1, the "lub"(components M1 and T1)|S1]]
*[[Heart sounds#Second heart tone S2 the "dub"(components A2 and P2)|S2]]
*[[Heart sounds#Third heart sound S3|S3]]
*[[Heart sounds#Fourth heart sound S4|S4]]
*[[Heart sounds#Summation Gallop|Gallops]]
*A high/low grade early/late [[systolic murmur]] / [[diastolic murmur]] best heard at the base/apex/(specific valve region) may be heard using the bell/diaphgram of the otoscope


===Abdomen===
===Abdomen===
Abdominal examination of patients with [disease name] is usually normal.
* Abdominal examination of patients with IgA nephropathy is usually normal.
 
OR
*[[Abdominal distention]]
*[[Abdominal tenderness]] in the right/left upper/lower abdominal quadrant
*[[Rebound tenderness]] (positive Blumberg sign)
*A palpable abdominal mass in the right/left upper/lower abdominal quadrant
*Guarding may be present
*[[Hepatomegaly]] / [[splenomegaly]] / [[hepatosplenomegaly]]
*Additional findings, such as obturator test, psoas test, McBurney point test, Murphy test


===Back===
===Back===
* Back examination of patients with [disease name] is usually normal.
* Back examination of patients with IgA nephropathy is usually normal.
OR
*Point tenderness over __ vertebrae (e.g. L3-L4)
*Sacral edema
*Costovertebral angle tenderness bilaterally/unilaterally
*Buffalo hump


===Genitourinary===
===Genitourinary===
* Genitourinary examination of patients with [disease name] is usually normal.
* Genitourinary examination of patients with IgA nephropathy is usually normal.
OR
*A pelvic/adnexal mass may be palpated
*Inflamed mucosa
*Clear/(color), foul-smelling/odorless penile/vaginal discharge


===Neuromuscular===
===Neuromuscular===
* Neuromuscular examination of patients with [disease name] is usually normal.
* Neuromuscular examination of patients with IgA nephropathy is usually normal.
OR
*Patient is usually oriented to persons, place, and time
* Altered mental status
* Glasgow coma scale is ___ / 15
* Clonus may be present
* Hyperreflexia / hyporeflexia / areflexia
* Positive (abnormal) Babinski / plantar reflex unilaterally/bilaterally
* Muscle rigidity
* Proximal/distal muscle weakness unilaterally/bilaterally
* ____ (finding) suggestive of cranial nerve ___ (roman numerical) deficit (e.g. Dilated pupils suggestive of CN III deficit)
*Unilateral/bilateral upper/lower extremity weakness
*Unilateral/bilateral sensory loss in the upper/lower extremity
*Positive straight leg raise test
*Abnormal gait (describe gait: e.g. ataxic (cerebellar) gait / steppage gait / waddling gait / choeiform gait / Parkinsonian gait / sensory gait)
*Positive/negative Trendelenburg sign
*Unilateral/bilateral tremor (describe tremor, e.g. at rest, pill-rolling)
*Normal finger-to-nose test / Dysmetria
*Absent/present dysdiadochokinesia (palm tapping test)


===Extremities===
===Extremities===
* Extremities examination of patients with [disease name] is usually normal.
*Pitting [[edema]] of the lower extremities develops in late stage, if the patient develops [[ESRD]]
OR
*[[Clubbing]]
*[[Cyanosis]]
*Pitting/non-pitting [[edema]] of the upper/lower extremities
*Muscle atrophy
*Fasciculations in the upper/lower extremity


==References==
==References==

Latest revision as of 13:40, 4 June 2018

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Dildar Hussain, MBBS [2]

Overview

Patients with IgA nephropathy usually appear normal and usually have no significant clinical finding upon physical examination. However, some of the patients may present with low-grade fever, high blood pressure with normal pulse pressure, and pitting edema of the lower extremities in the late stage if the patient develops ESRD.

Physical Examination

  • Physical examination of patients with IgA nephropathy is usually normal.[1][2][3][4][5]

Appearance of the Patient

  • Patients with IgA nephropathy usually appear normal.

Vital Signs

Skin

  • Skin examination of patients with IgA nephropathy is usually normal.

HEENT

  • HEENT examination of patients with IgA nephropathy is usually normal.

Neck

  • Neck examination of patients with IgA nephropathy is usually normal.

Lungs

  • Pulmonary examination of patients with IgA nephropathy is usually normal.

Heart

  • Cardiovascular examination of patients with IgA nephropathy is usually normal.

Abdomen

  • Abdominal examination of patients with IgA nephropathy is usually normal.

Back

  • Back examination of patients with IgA nephropathy is usually normal.

Genitourinary

  • Genitourinary examination of patients with IgA nephropathy is usually normal.

Neuromuscular

  • Neuromuscular examination of patients with IgA nephropathy is usually normal.

Extremities

  • Pitting edema of the lower extremities develops in late stage, if the patient develops ESRD

References

  1. Galla JH (February 1995). "IgA nephropathy". Kidney Int. 47 (2): 377–87. PMID 7723227.
  2. Donadio JV, Grande JP (September 2002). "IgA nephropathy". N. Engl. J. Med. 347 (10): 738–48. doi:10.1056/NEJMra020109. PMID 12213946.
  3. Hall CL, Bradley R, Kerr A, Attoti R, Peat D (October 2004). "Clinical value of renal biopsy in patients with asymptomatic microscopic hematuria with and without low-grade proteinuria". Clin. Nephrol. 62 (4): 267–72. PMID 15524056.
  4. Topham PS, Harper SJ, Furness PN, Harris KP, Walls J, Feehally J (June 1994). "Glomerular disease as a cause of isolated microscopic haematuria". Q. J. Med. 87 (6): 329–35. PMID 8041865.
  5. Gutierrez, E.; Gonzalez, E.; Hernandez, E.; Morales, E.; Martinez, M. A.; Usera, G.; Praga, M. (2006). "Factors That Determine an Incomplete Recovery of Renal Function in Macrohematuria-Induced Acute Renal Failure of IgA Nephropathy". Clinical Journal of the American Society of Nephrology. 2 (1): 51–57. doi:10.2215/CJN.02670706. ISSN 1555-9041.

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