Sandbox: jss: Difference between revisions

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{| border="1"
{{familytree/start |summary=PE diagnosis Algorithm.}}
|+ The table's caption
{{familytree | | | | | | | | | | A01 | | | | |A01=Serum Potassium >5.1meq/L}}
! Differentiating features !! Leukemia !! Leukemoid reaction
{{familytree | | | | | | | | | | |!| | | | | | | }}
|-
{{familytree | | | | | | | | | | A02 | | | | | |A02=Measure GFR}}
! Causes
{{familytree | | | |,|-|-|-|-|-|-|^|-|-|-|-|-|-|-|.| | | | | | }}
| Carcinogens || Infectious agents, biologically active substances and
{{familytree | | | B01 | | | | | | | | | | | | | B02 | | |B01=GFR <15ml/min/1.73m2|B02=GFR>15ml/min/1.73m2}}
products of tissue destruction
{{familytree | | | |!| | | | | | | | | | | | | | |!| | | | }}
|-
{{familytree | | | C01 | | | | | | | | | | | | | |!| |C01=Measure urinary potassium|C02=Measure Aldosterone}}
! Pathogenesis
{{familytree | |,|-|^|-|.| | | | | | | | |,|-|-|-|+|-|-|-|.| | }}
| The transformation of normal haematopoietic
{{familytree | |!| | | |!| | | | | | | | |!| | | |!| | | |!| | }}
cells to a tumor
{{familytree | D01 | | D02 | | | | | | | D03| | |D04 | | D05| |D01=Urine potassium <20meq/L|D02=urine potassium >125meq/L|D03=Aldosterone low|D04=Aldosterone normal\high}}
| Activation of normal hematopoiesis and exit of immature leukocytes
{{familytree | |!| | | |!| | | | | | | | |!| | | |!| | | | }}
into the bloodstream. 
{{familytree | E01 | | E02 | | | | | | | E03 | | E04 | | | |E01=*Acute renal failure<br>Chronic renal failure<br>Heart failure<br>Volume depletion<br>Tissue breakdown<br>*Blood transfusion|E02=Supplemental potassium|E03=Measure renin|E04=Pseudohypoaldostrenism type 1 and type 2<br>Aldosterone receptor antagonist<br>Amiloride<br>Sprironolactone<br>Type 1 RTA }}
|-
{{familytree | | | | | |!| | | | | | | | | | |!| | | |!| | | | }}
! Duration
{{familytree | | | | | | | | | | | | | | | | |!| | }}
| Chronic
{{familytree | | | |,|-|^|-|.| | | | | | |,|-|^|-|.| |!| | }}
| Temporary and reversible
{{familytree | | | G01 | | G02 | | | | | G03 | | | | G04 |!| | |G01=G01|G02=G02|G03=Renin low|G04=Renin normal/high}}
|-
{{familytree | | | |!| | | |!| | | | | | |!| | | |,|-|^|-|v|-|-|.|}}
! Genetic level
{{familytree | | | H01 | | H02 | | | | | H03 | | H04 | | H05 | | H06 |H01=H01|H02=H02|H03=Hyporenimic Hypoaldosteronism|H04=Primary hypoaldosteronism<br>Congenital adrenal hyperplasia<br>Secondary hypoaldosteronism<br>Drugs|H05=High serum osmolarity<br>Diabteic Ketoacidosis|H06=Metabloic acidosis}}
| Defect
{{familytree | | | |!| | | | | | | | | | |!| | | |!| | | | | }}
| No defect
{{familytree | | | I01 | | | | | | | | | I02 | | I03 | | | |I01=I01|I02=Interstitial nephritis<br>Obstructive uropathy<br>ACE inhibitors<Angiotensin 2 receptor antagonists |I03=}}
|-
{{familytree | |,|-|^|-|.| | | | | | | | |!| |,|-|^|-|.| | | |}}
! Splenomegaly
{{familytree | J01 | | J02 | | | | | | | |!| J03 | | J04 | | | |J01=J01|J02=J02|J03=J03|J04=J04}}
| Present
{{familytree | |!| | | | | | | | | | | | |!| |!| | | |!| | |}}
| Not present
{{familytree | K01 |-|-|-|-|-|-|-|-|-|-|-|'| |!| | | K02 | |K01=K01|K02=K02}}
|-
{{familytree | |!| | | | | | | | | | | | | | |!| | | | | | |}}
! Peripheral blood
{{familytree | |`|-|-|-|-|-|-|-|-|-|-|-|-|-|-|'| | | | | | |}}
| Immature cells, pancytopenia
{{familytree/end}}
| Mature and immature granulocytes with left shift
|-
! Bone marrow
| Increase in blasts and immature cells
| Myeloid hyperplasia and normal morphology
|-
! LAP score
| Low
| High
|-
! Toxic granules
(suggestive of infection)
| Absent
| Present
|-
! Dohle inclusion bodies
| Absent
| Present

Latest revision as of 14:25, 9 October 2018


Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]


The table's caption
Differentiating features Leukemia Leukemoid reaction
Causes Carcinogens Infectious agents, biologically active substances and

products of tissue destruction

Pathogenesis The transformation of normal haematopoietic

cells to a tumor

Activation of normal hematopoiesis and exit of immature leukocytes

into the bloodstream.

Duration Chronic Temporary and reversible
Genetic level Defect No defect
Splenomegaly Present Not present
Peripheral blood Immature cells, pancytopenia Mature and immature granulocytes with left shift
Bone marrow Increase in blasts and immature cells Myeloid hyperplasia and normal morphology
LAP score Low High
Toxic granules 
(suggestive of infection)
Absent Present
Dohle inclusion bodies Absent Present
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