Sandbox: jss: Difference between revisions

Latest revision as of 14:25, 9 October 2018

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

The table's caption
Differentiating features	Leukemia	Leukemoid reaction
Causes	Carcinogens	Infectious agents, biologically active substances and products of tissue destruction
Pathogenesis	The transformation of normal haematopoietic cells to a tumor	Activation of normal hematopoiesis and exit of immature leukocytes into the bloodstream.
Duration	Chronic	Temporary and reversible
Genetic level	Defect	No defect
Splenomegaly	Present	Not present
Peripheral blood	Immature cells, pancytopenia	Mature and immature granulocytes with left shift
Bone marrow	Increase in blasts and immature cells	Myeloid hyperplasia and normal morphology
LAP score	Low	High
Toxic granules (suggestive of infection)	Absent	Present
Dohle inclusion bodies	Absent	Present

@@ Line 4: / Line 4: @@
-{{Family tree/start}}
+{| border="1"
-{{Family tree | | | | | | | | | | | | | | A01 | | | | | | | | | | | |A01=Potassium >5.1meq/L}}
+|+ The table's caption
-{{Family tree | | | | | | | | | | | | | | |!| | | | | | | | | | | | }}
+! Differentiating features !! Leukemia !! Leukemoid reaction
-{{Family tree | | | | | | | | | | | | | | B01 | | | | | | | | | | | B01= ECG }}
+|-
-{{Family tree | | | | | | | | | | | | |,|-|^|-|.| | | | | | | | | | }}
+! Causes
-{{Family tree | | | | | | | | | | | | C01 | | C02 | | | | | | | | | C01=If no changes,rule out pseudohyperkalemia| C02= If changes present then start urgent treatment}}
+| Carcinogens || Infectious agents, biologically active substances and
-{{Family tree | | | | | | | |,|-|-|-|-|^|-|-|-|-|.| | | | | | | | |}}
+products of tissue destruction
-{{Family tree | | | | | | | D01 | | | | | | | D02 |D01=Urine sodium <25 meq/L|D02=urine sodium >25 meq/L }}
+|-
-{{Family tree | | | | | | | |!| | | |,|-|-|-|-|^|-|-|-|-|.| | | | | }}
+! Pathogenesis
-{{Family tree | | | | | | | E01 | | E02 | | | | | | | | E03 | | | | | | | E01=ARF<br>CKD<br>Heart failure<br>Volume depletion|E02=Decreased K+secretion(Urine K+<20meq/L|E03=Transcellular shift(measure serum osmolarity and pH) }}
+| The transformation of normal haematopoietic
-{{familytree  | | | | | | |,|-|-|-|-|^|-|-|.| | | | | | |!| | | | | | | | }}
+cells to a tumor
-{{Family tree | | | | | | F01 | | | | | | F02 | | | | | F03 | | | | | | F01=Low aldosterone(TTG<3)|F02=Normal aldosterone(TTG>7)|F03=Diabetic ketoacidosis<br>Metabolic acidosis}}
+| Activation of normal hematopoiesis and exit of immature leukocytes
-{{Family tree | | | | |,|-|^|-|.| | | | | |!| | | | | | | | | | | }}
+into the bloodstream.
-{{Family tree | | | | G01 | | G02 | | | | G03 | | | | | | | | | | G01=Low renin|G02=Normal renin|G03=Tissue breakdown<br>Pseudohypoaldosternism type 1 and type 2<br>Type 1 RTA }}
+|-
-{{Family tree | | | | |!| | | |!| | | | | | | | | | | | }}
+! Duration
-{{Family tree | | | | H01 | | H02 | | | | | | | | | H01=Interstital nephritis<br>Obstructive uropathies<br>Diabetic nephropathy<br>ACE inhibitors,Angiotensin 2 receptors|H02=Primary hypoaldosteronism<br>Congenital adrenal hyperplasia<br>Aldosterone receptor antagonists<br>RTA type 4
+| Chronic
-}}
+| Temporary and reversible
+|-
+! Genetic level
+| Defect
+| No defect
+|-
+! Splenomegaly
+| Present
+| Not present
+|-
+! Peripheral blood
+| Immature cells, pancytopenia
+| Mature and immature granulocytes with left shift
+|-
+! Bone marrow
+| Increase in blasts and immature cells
+| Myeloid hyperplasia and normal morphology
+|-
+! LAP score
+| Low
+| High
+|-
+! Toxic granules
+ (suggestive of infection)
+| Absent
+| Present
+|-
+! Dohle inclusion bodies
+| Absent
+| Present

Sandbox: jss: Difference between revisions

Latest revision as of 14:25, 9 October 2018

Navigation menu