Asystole resident survival guide: Difference between revisions

Jump to navigation Jump to search
m (Bot: Removing from Primary care)
 
(25 intermediate revisions by 4 users not shown)
Line 2: Line 2:
{{CMG}}; {{AE}} {{MS}}
{{CMG}}; {{AE}} {{MS}}


==Definition==
==Overview==
Asystole is a state of no [[heart|cardiac]] electrical activity, hence no contractions of the [[myocardium]] and no cardiac output or blood flow.  Asystole is also known as a cardiac arrest rhythm in which there is no distinct electrical activity on ECG.  A (flat line) is another acronym for asystole. In asystole, the heart will not respond to [[defibrillation]] because it is already [[Depolarization|depolarized]].
Asystole is a state of no [[heart|cardiac]] electrical activity, hence no contractions of the [[myocardium]] and no cardiac output or blood flow.  Asystole is also known as a cardiac arrest rhythm in which there is no distinct electrical activity on ECG.  A (flat line) is another acronym for asystole. In asystole, the heart will not respond to [[defibrillation]] because it is already [[Depolarization|depolarized]].


Line 26: Line 26:


==Management==
==Management==
Below is an algorithm summarizing the approach to a patient with asystole. Based on the 2010 American heart association ACLS algorithm for asystole
Below is an algorithm summarizing the approach to a patient with asystole. Based on the 2010 American heart association ACLS algorithm for asystole<ref name="pmid20956217">{{cite journal| author=Field JM, Hazinski MF, Sayre MR, Chameides L, Schexnayder SM, Hemphill R et al.| title=Part 1: executive summary: 2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. | journal=Circulation | year= 2010 | volume= 122 | issue= 18 Suppl 3 | pages= S640-56 | pmid=20956217 | doi=10.1161/CIRCULATIONAHA.110.970889 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=20956217  }} </ref>


{{familytree/start}}
{{familytree/start}}
{{familytree | | | | | | | | | A01 | | | | | |A01=Asystole<br>[[Image:Lead II rhythm generated asystole.JPG|350px|left|thumb]]}}
{{familytree | | | | | | | | | | | | | | |A01 | | | | | |A01=Asystole<br>[[Image:Lead II rhythm generated asystole.JPG|350px|left|thumb]]}}
{{familytree | | | | | | | | | |!| | | | | | | | }}
{{familytree | | | | | | | | | | | | | | | |!| | | | | | | | }}
{{familytree | | | | | | | | | B01 | | | | | |B01=Start CPR for 2 minutes<br>Give oxygen<br>Attach monitor and defibrillator<br>IV/IO access<br>Epinephrine Q3-5 min<br>Consider advanced airway, capnography}}
{{familytree | | | | | | | | | | | | | | |B01 | | | | | |B01=Start CPR for 2 minutes<br>Give oxygen<br>Attach monitor and defibrillator<br>IV/IO access<br>Epinephrine Q3-5 min<br>Consider advanced airway, capnography}}
{{familytree | | | | | | | | | |!| | | | | | | | }}
{{familytree | | | | | | | | | | | | | | | |!|| | | | | | | | }}
{{familytree | | | | | | | | | C01 | | | | | |C01=Rhythm}}
{{familytree | | | | | | | | | | | | | | |C01 | | | | | |C01=Rhythm}}
{{familytree | | |,|-|-|-|-|-|-|^|-|-|-|-|-|-|.| }}
{{familytree | | |,|-|-|-|-|-|-|-|-|-|-|-|-|^|-|-|-|-|-|-|.| }}
{{familytree | | C01 | | | | | | | | | | | |C02|C01=Shockable|C02=Non-shockable}}
{{familytree | | C01 | | | | | | | | | | | | | | | | | |C02|C01=Shockable|C02=Non-shockable}}
{{familytree | | |!| | | | | | | | | | | | | |!| | | | | | | | }}
{{familytree | | |!| | | | | | | | | | | | | | | | | | | |!| | | | | | | | }}
{{familytree | |D01| | | | | | | | | | | |D02|D01=See VF/VT algorithm|D02=CPR for 2 minutes<br>Treat Hs&Ts}}
{{familytree | |D01| | | | | | | | | | | | | | | | | |D02|-|-|-|-|-|-|-|.| |D01=See VF/VT algorithm|D02=CPR for 2 minutes<br>Treat Hs&Ts<br>Epinephrine Q3-5min}}
{{familytree | | | | | | | | | | | | | | | | |!| | | | | | | | }}
{{familytree | | |!| | | | | | | | | | | | | | | | | | | |!| | | | | | | | |!| | | | | | | | | | | | | | |}}
{{familytree | | | | | | | | | | | | | | | | C01 | | | | | |C01=Rhythm}}
{{familytree | | |!| | | | | | | | | | | | | | | | | | | C01 | | | | | | | |!| | | | | | | | | | | | | | |C01=Rhythm}}
{{familytree | | | | | | | | | | |,|-|-|-|-|-|^|-|-|-|-|-|-|.| }}
{{familytree | | |!| | | | | | | | |,|-|-|-|-|-|-|-|-|-|-|^|-|-|-|-|.| | | |!| | | | | | | | |}}
{{familytree | | | | | | | | | | |C1| | | | | | | | | |C02|C1=Shockable|C02=Non-shockable}}
{{familytree | | |`|-|-|-|-|-|-|-|C1 | | | | | | | | | | | | | |C02|-|-|'| | | | | | |C1=Shockable|C02=Non-shockable}}
{{familytree | | | | | | | | | | | |!| | | | | | | | | | | | | | | |!| | }}
{{familytree | | | | | | | | | | |Z|-|-|-|-|-|-|-|-|-|-|-|-|-|-|'| | | | |Z=ROSC(return of spontaneous circulation}}
{{familytree | | | | | | | | | | | |!| | | | | | | | }}
{{familytree | | | | | | | | | | |Z1| | | | | | | | |Z1=Post–Cardiac Arrest Care }}
{{familytree/end}}
{{familytree/end}}


==Do's==
==Do's==
* Efficiency of CPR can be determined by
**Monitoring of chest compression rate and depth
**Adequacy of chest wall relaxation
**Length and duration of pauses in compression and number and depth of ventilations delivered
** Physiologic parameters; partial pressure of end-tidal CO2 [PETCO2], arterial pressure during the relaxation phase of chest compressions, central venous oxygen saturation [ScvO2]
* Remember that the foundation of successful ACLS is good BLS , represented in prompt high-quality CPR with minimal interruptions.<ref name="pmid16982127">{{cite journal| author=Edelson DP, Abella BS, Kramer-Johansen J, Wik L, Myklebust H, Barry AM et al.| title=Effects of compression depth and pre-shock pauses predict defibrillation failure during cardiac arrest. | journal=Resuscitation | year= 2006 | volume= 71 | issue= 2 | pages= 137-45 | pmid=16982127 | doi=10.1016/j.resuscitation.2006.04.008 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=16982127  }} </ref><ref name="pmid12010909">{{cite journal| author=Eftestøl T, Sunde K, Steen PA| title=Effects of interrupting precordial compressions on the calculated probability of defibrillation success during out-of-hospital cardiac arrest. | journal=Circulation | year= 2002 | volume= 105 | issue= 19 | pages= 2270-3 | pmid=12010909 | doi= | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=12010909  }} </ref>
* A new class I recommendation is the use of quantitative waveform capnography for confirmation and monitoring of endotracheal tube placement.
*  Supraglottic advanced airways continues to be an alternative to endotracheal intubation for airway management during CPR.


==Don'ts==
==Don'ts==
* Don't routinely use cricoid pressure during airway management of patients in cardiac arrest.
* Don't routinely administer atropine in the management of pulseless asystole.


==References==
==References==
{{Reflist|2}}
{{Reflist|2}}
{{WikiDoc Help Menu}}
{{WikiDoc Sources}}
[[CME Category::Cardiology]]


[[Category:Disease]]
[[Category:Disease]]
[[Category:Emergency medicine]]
[[Category:Emergency medicine]]
[[Category:Medicine]]
[[Category:Medicine]]
[[Category:Primary care]]
[[Category:Resident survival guide]]
[[Category:Resident survival guide]]
[[Category:Signs and symptoms]]
[[Category:Cardiology]]
[[Category:Cardiology]]
[[Category:Up-To-Date]]
[[Category:Up-To-Date]]
[[Category:Up-To-Date cardiology]]
[[Category:Up-To-Date cardiology]]
[[Category:Intensive care medicine]]
[[Category:Intensive care medicine]]
{{WikiDoc Help Menu}}
{{WikiDoc Sources}}

Latest revision as of 20:31, 29 July 2020

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Mahmoud Sakr, M.D. [2]

Overview

Asystole is a state of no cardiac electrical activity, hence no contractions of the myocardium and no cardiac output or blood flow. Asystole is also known as a cardiac arrest rhythm in which there is no distinct electrical activity on ECG. A (flat line) is another acronym for asystole. In asystole, the heart will not respond to defibrillation because it is already depolarized.

Causes

Life Threatening Causes

Asystole is a life-threatening condition and must be treated as such irrespective of the causes. Life-threatening conditions can result in death or permanent disability within 24 hours if left untreated.

Common Causes

Management

Below is an algorithm summarizing the approach to a patient with asystole. Based on the 2010 American heart association ACLS algorithm for asystole[1]

 
 
 
 
 
 
 
 
 
 
 
 
 
 
Asystole
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Start CPR for 2 minutes
Give oxygen
Attach monitor and defibrillator
IV/IO access
Epinephrine Q3-5 min
Consider advanced airway, capnography
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Rhythm
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Shockable
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Non-shockable
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
See VF/VT algorithm
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
CPR for 2 minutes
Treat Hs&Ts
Epinephrine Q3-5min
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Rhythm
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Shockable
 
 
 
 
 
 
 
 
 
 
 
 
 
Non-shockable
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
ROSC(return of spontaneous circulation
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Post–Cardiac Arrest Care
 
 
 
 
 
 
 
 

Do's

  • Efficiency of CPR can be determined by
    • Monitoring of chest compression rate and depth
    • Adequacy of chest wall relaxation
    • Length and duration of pauses in compression and number and depth of ventilations delivered
    • Physiologic parameters; partial pressure of end-tidal CO2 [PETCO2], arterial pressure during the relaxation phase of chest compressions, central venous oxygen saturation [ScvO2]
  • Remember that the foundation of successful ACLS is good BLS , represented in prompt high-quality CPR with minimal interruptions.[2][3]
  • A new class I recommendation is the use of quantitative waveform capnography for confirmation and monitoring of endotracheal tube placement.
  • Supraglottic advanced airways continues to be an alternative to endotracheal intubation for airway management during CPR.

Don'ts

  • Don't routinely use cricoid pressure during airway management of patients in cardiac arrest.
  • Don't routinely administer atropine in the management of pulseless asystole.

References

  1. Field JM, Hazinski MF, Sayre MR, Chameides L, Schexnayder SM, Hemphill R; et al. (2010). "Part 1: executive summary: 2010 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care". Circulation. 122 (18 Suppl 3): S640–56. doi:10.1161/CIRCULATIONAHA.110.970889. PMID 20956217.
  2. Edelson DP, Abella BS, Kramer-Johansen J, Wik L, Myklebust H, Barry AM; et al. (2006). "Effects of compression depth and pre-shock pauses predict defibrillation failure during cardiac arrest". Resuscitation. 71 (2): 137–45. doi:10.1016/j.resuscitation.2006.04.008. PMID 16982127.
  3. Eftestøl T, Sunde K, Steen PA (2002). "Effects of interrupting precordial compressions on the calculated probability of defibrillation success during out-of-hospital cardiac arrest". Circulation. 105 (19): 2270–3. PMID 12010909.


Template:WikiDoc Sources CME Category::Cardiology