Carotid body tumor risk factors: Difference between revisions
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{{Carotid body tumor}} | {{Carotid body tumor}} | ||
{{CMG}}; {{AE}} {{Sahar}} | {{CMG}}; {{AE}} {{Sahar}} {{MV}} | ||
==Overview== | ==Overview== | ||
Common [[risk factors]] in the development of [[carotid body]] [[tumor]] include [[genetic mutation]] and [[chronic]] [[hypoxia]]. | |||
==Risk Factors== | ==Risk Factors== | ||
*Common risk factors in the development of carotid body tumor, include:<ref name=" | *Common [[risk factors]] in the development of [[carotid body]] [[tumor]], include:<ref name="WienekeSmith2009">{{cite journal|last1=Wieneke|first1=Jacqueline A.|last2=Smith|first2=Alice|title=Paraganglioma: Carotid Body Tumor|journal=Head and Neck Pathology|volume=3|issue=4|year=2009|pages=303–306|issn=1936-055X|doi=10.1007/s12105-009-0130-5}}</ref> | ||
:*Genetic mutations in the following genes: | :*[[Genetic mutations]] in the following [[genes]]: | ||
::*MEN 2A | ::*[[MEN 2a|MEN 2A]] | ||
::*MEN 2B | ::*[[MEN 2B]] | ||
::* | ::*[[RET gene|RET]] | ||
::*[[VHL]] | |||
::*[[NF1]] | |||
::*[[SDH]] subunits ([[SDHD]], [[SDHB]], and [[SDHC]]) | |||
*[[Chronic]] [[hypoxia]] has been observed to be associated with [[hyperplastic]] form of [[carotid body]] [[tumor]].<ref name="BurgessCalderon2017">{{cite journal|last1=Burgess|first1=Alfred|last2=Calderon|first2=Moises|last3=Jafif-Cojab|first3=Marcos|last4=Jorge|first4=Diego|last5=Balanza|first5=Ricardo|title=Bilateral carotid body tumor resection in a female patient|journal=International Journal of Surgery Case Reports|volume=41|year=2017|pages=387–391|issn=22102612|doi=10.1016/j.ijscr.2017.11.019}}</ref><ref name="SajidHamilton2007">{{cite journal|last1=Sajid|first1=M.S.|last2=Hamilton|first2=G.|last3=Baker|first3=D.M.|title=A Multicenter Review of Carotid Body Tumour Management|journal=European Journal of Vascular and Endovascular Surgery|volume=34|issue=2|year=2007|pages=127–130|issn=10785884|doi=10.1016/j.ejvs.2007.01.015}}</ref> | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
[[Category:Medicine]] | [[Category:Medicine]] | ||
[[Category:Oncology]] | [[Category:Oncology]] | ||
[[Category: | [[Category:Up-To-Date]] | ||
[[Category:Surgery]] | [[Category:Surgery]] |
Latest revision as of 20:49, 29 July 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Sahar Memar Montazerin, M.D.[2] Maria Fernanda Villarreal, M.D. [3]
Overview
Common risk factors in the development of carotid body tumor include genetic mutation and chronic hypoxia.
Risk Factors
- Common risk factors in the development of carotid body tumor, include:[1]
- Genetic mutations in the following genes:
- Chronic hypoxia has been observed to be associated with hyperplastic form of carotid body tumor.[2][3]
References
- ↑ Wieneke, Jacqueline A.; Smith, Alice (2009). "Paraganglioma: Carotid Body Tumor". Head and Neck Pathology. 3 (4): 303–306. doi:10.1007/s12105-009-0130-5. ISSN 1936-055X.
- ↑ Burgess, Alfred; Calderon, Moises; Jafif-Cojab, Marcos; Jorge, Diego; Balanza, Ricardo (2017). "Bilateral carotid body tumor resection in a female patient". International Journal of Surgery Case Reports. 41: 387–391. doi:10.1016/j.ijscr.2017.11.019. ISSN 2210-2612.
- ↑ Sajid, M.S.; Hamilton, G.; Baker, D.M. (2007). "A Multicenter Review of Carotid Body Tumour Management". European Journal of Vascular and Endovascular Surgery. 34 (2): 127–130. doi:10.1016/j.ejvs.2007.01.015. ISSN 1078-5884.