Hay fever pathophysiology: Difference between revisions
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[[Category:Allergology]] | [[Category:Allergology]] | ||
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Latest revision as of 21:58, 29 July 2020
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Hay fever Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Hay fever pathophysiology On the Web |
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American Roentgen Ray Society Images of Hay fever pathophysiology |
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Risk calculators and risk factors for Hay fever pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Pathophysiology
As noted above, hay fever involves an allergic reaction to pollen. A virtually identical reaction occurs with allergy to mold, animal dander, dust and similar inhaled allergens. Particulate matter in polluted air and chemicals such as chlorine and detergents, which can normally be tolerated, can greatly aggravate the condition.
When an allergen such as pollen or dust is inhaled by a person with a sensitized immune system, it triggers antibody production. These antibodies mostly bind to mast cells, which contain histamine. When the mast cells are stimulated by pollen and dust, histamine (and other chemicals) are released. This causes itching, swelling, and mucus production. Symptoms vary in severity from person to person. Very sensitive individuals can experience hives or other rashes.
Some disorders may be associated with allergies, which are common. Heredity and environmental exposures may contribute to a predisposition to allergies. These include eczema and asthma, among others.
The time of year at which hay fever symptoms manifest themselves varies greatly depending on the types of pollen to which an allergic reaction is produced. The pollen count, in general, is highest from mid-spring to early summer. As most pollens are produced at fixed periods in the year, a long-term hay fever sufferer may also be able to anticipate when the symptoms are most likely to begin and end, although this may be complicated by an allergy to dust particles.