Polycystic kidney disease laboratory findings: Difference between revisions
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{{Polycystic kidney disease}} | {{Polycystic kidney disease}} | ||
{{CMG}} {{AE}} [[User:Sergekorjian|Serge Korjian]],[[User:YazanDaaboul|Yazan Daaboul]] | {{CMG}}; {{AE}}{{MKA}}, [[User:Sergekorjian|Serge Korjian]],[[User:YazanDaaboul|Yazan Daaboul]] | ||
==Overview== | ==Overview== | ||
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==Laboratory Findings== | ==Laboratory Findings== | ||
Laboratory findings in polycystic kidney disease are usually confined to [[urinary]] abnormalities, which include:<ref name="pmid8198379">{{cite journal| author=Fick GM, Gabow PA| title=Natural history of autosomal dominant polycystic kidney disease. | journal=Annu Rev Med | year= 1994 | volume= 45 | issue= | pages= 23-9 | pmid=8198379 | doi=10.1146/annurev.med.45.1.23 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=8198379 }} </ref> | |||
* Reduction in maximal [[urine]] [[concentration]] after water deprivation and [[ADH]] administration (usually first manifestation of disease) | * Reduction in maximal [[urine]] [[concentration]] after water deprivation and [[ADH]] administration (usually first manifestation of disease) | ||
* [[Hypocitraturia]] in 65% on patients | * [[Hypocitraturia]] in 65% on patients | ||
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{{WH}} | {{WH}} | ||
{{WS}} | {{WS}} | ||
[[Category:Up-To-Date]] | |||
[[Category:Medicine]] | |||
[[Category:Nephrology]] |
Latest revision as of 23:46, 29 July 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: M. Khurram Afzal, MD [2], Serge Korjian,Yazan Daaboul
Overview
Common laboratory findings in polycystic kidney disease are urinary abnormalities including reduction in concentration capacity, hypocitraturia, hematuria, and proteinuria. Hyperuricemia is also a common finding and is a risk factor for disease progression and ESRD.
Laboratory Findings
Laboratory findings in polycystic kidney disease are usually confined to urinary abnormalities, which include:[1]
- Reduction in maximal urine concentration after water deprivation and ADH administration (usually first manifestation of disease)
- Hypocitraturia in 65% on patients
- Hyperuricemia in 20% of patients
- Hyperoxaluria in 20% of patients
- Low urine pH
- Hematuria (microscopic or macroscopic)
- Proteinuria usually less than 1 g/day
References
- ↑ Fick GM, Gabow PA (1994). "Natural history of autosomal dominant polycystic kidney disease". Annu Rev Med. 45: 23–9. doi:10.1146/annurev.med.45.1.23. PMID 8198379.