Dermatitis herpetiformis risk factors: Difference between revisions
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==Overview== | ==Overview== | ||
Dermatitis | Dermatitis Herpetiformis (also called [[Duhring's disease]]), is a chronic itchy rash which is frequently associated with [[Celiac Disease]]. The rash is made of [[papules]] and [[vesicles]] which are present on different parts of the body mostly commonly on neck, trunk, buttocks and knees. It is an [[autoimmune]] mediated skin condition, which is IgA mediated reaction and is associated with [[gluten sensitivity]] of small bowel. There is presence of [[antibodies]] which leads to positive serology test results. | ||
Dermatitis herpetiformis is associated with high prevalence of other [[autoimmune diseases]]. | |||
==Risk Factors== | ==Risk Factors== | ||
The most important risk factor in the development of this rash of dermatitis herpetiformis is presence of [[Gluten]] in the diet of genetically susceptible individuals. see [[Dermatitis herpetiformis pathophysiology]] | |||
==References== | ==References== |
Latest revision as of 00:56, 7 February 2021
Dermatitis herpetiformis Microchapters |
Differentiating Dermatitis herpetiformis from other Diseases |
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Dermatitis herpetiformis risk factors On the Web |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Dermatitis Herpetiformis (also called Duhring's disease), is a chronic itchy rash which is frequently associated with Celiac Disease. The rash is made of papules and vesicles which are present on different parts of the body mostly commonly on neck, trunk, buttocks and knees. It is an autoimmune mediated skin condition, which is IgA mediated reaction and is associated with gluten sensitivity of small bowel. There is presence of antibodies which leads to positive serology test results.
Dermatitis herpetiformis is associated with high prevalence of other autoimmune diseases.
Risk Factors
The most important risk factor in the development of this rash of dermatitis herpetiformis is presence of Gluten in the diet of genetically susceptible individuals. see Dermatitis herpetiformis pathophysiology