Ketoacidosis pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
Ketoacidosis occurs when cells do not have sufficient glucose to meet their metabolic demands. Instead, [[ketone bodies]] are produced for energy via the [[metabolism]] of [[fatty acids]]. Acidity results from the dissociation of the H<sup>+</sup> ion at physiological pH of metabolic ketone bodies such as [[acetoacetate]], and [[Beta-hydroxybutyrate|β-hydroxybutyrate]]. | |||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
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[[Category:Needs content]] | |||
[[Category:Medical emergencies]] | [[Category:Medical emergencies]] | ||
[[Category:Endocrinology]] | [[Category:Endocrinology]] | ||
Latest revision as of 17:02, 13 February 2013
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Ketoacidosis Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Ketoacidosis pathophysiology On the Web |
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American Roentgen Ray Society Images of Ketoacidosis pathophysiology |
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Risk calculators and risk factors for Ketoacidosis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Overview
Ketoacidosis occurs when cells do not have sufficient glucose to meet their metabolic demands. Instead, ketone bodies are produced for energy via the metabolism of fatty acids. Acidity results from the dissociation of the H+ ion at physiological pH of metabolic ketone bodies such as acetoacetate, and β-hydroxybutyrate.