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| __NOTOC__
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| {{Suicidal ideation}}
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| {{CMG}}; {{AE}}
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| ==Overview==
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| The exact pathogenesis of [disease name] is not fully understood.
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| OR
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| It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
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| OR
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| [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
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| OR
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| Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
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| OR
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| [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
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| OR
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| The progression to [disease name] usually involves the [molecular pathway].
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| OR
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| The pathophysiology of [disease/malignancy] depends on the histological subtype.
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| ==Pathophysiology==
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| Decreased cerebrospinal fluid (CSF) serotonin levels and serotonin receptor dysfunction are strong risk factors for suicide attempt and completion.
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| ==Genetics==
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| *[Disease name] is transmitted in [mode of genetic transmission] pattern.
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| *Genes involved in the pathogenesis of [disease name] include [gene1], [gene2], and [gene3].
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| *The development of [disease name] is the result of multiple genetic mutations.
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| ==Associated Conditions==
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| ==Gross Pathology==
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| *On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
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| ==Microscopic Pathology==
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| *On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
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| ==References==
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| {{Reflist|2}}
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| {{WH}}
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| {{WS}}
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