Hyperkalemia risk factors: Difference between revisions

	Hyperkalemia Microchapters
Home
Patient information
Overview
Historical Perspective
Classification
Pathophysiology
Causes
Differentiating Hyperkalemia from other Diseases
Epidemiology and Demographics
Risk Factors
Screening
Natural history, Complications and Prognosis
Diagnosis
Diagnostic Study of Choice
History and Symptoms
Physical Examination
Laboratory Findings
Electrocardiogram
X-ray
Echocardiography and Ultrasound
CT scan
MRI
Other Imaging Findings
Other Diagnostic Studies
Treatment
Medical Therapy
Surgery
Primary Prevention
Secondary Prevention
Cost-Effectiveness of Therapy
Future or Investigational Therapies
Case Studies
Case #1
Hyperkalemia risk factors On the Web
Most recent articles
Most cited articles
Review articles
CME Programs
Powerpoint slides
Images
American Roentgen Ray Society Images of Hyperkalemia risk factors All Images X-rays Echo & Ultrasound CT Images MRI
Ongoing Trials at Clinical Trials.gov
US National Guidelines Clearinghouse
NICE Guidance
FDA on Hyperkalemia risk factors
CDC on Hyperkalemia risk factors
Hyperkalemia risk factors in the news
Blogs on Hyperkalemia risk factors
Directions to Hospitals Treating Hyperkalemia
Risk calculators and risk factors for Hyperkalemia risk factors

VisualWikitext

Latest revision as of 23:04, 29 April 2020

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-In-Chief: Priyamvada Singh, M.B.B.S. [2],Jogeet Singh Sekhon

Overview

The kidneys normally remove excess potassium from the body. Most cases of hyperkalemia occur in disorders that reduce the kidneys' ability to get rid of potassium. This may result from disorders such as acute kidney failure, chronic kidney failure and glomerulonephritis.

Risk Factors

The kidneys normally remove excess potassium from the body
Most cases of hyperkalemia are due to disorders that reduce the kidneys' ability to get rid of potassium ^[1]^[2]

Common risk factors

The most common risk factors for hyperkalmeia include^[3]:
- Acute kidney failure
- Chronic kidney failure^[4]
- Diabetes mellitus
- Glomerulonephritis
- Obstructive uropathy
- Rejection of a kidney transplant
- Addison's disease

Burns
Disorders that cause blood cells to burst (hemolytic conditions)
Gastrointestinal bleeding
Rhabdomyolysis from drugs, alcoholism, coma, or certain infections
Surgery
Traumatic injury
Tumors
Acidosis
Medications
- spironolactone
- amiloride
- triamterene
- Potassium supplements (especially intravenous potassium).

Less common risk factors

References

↑ Wang WH, Giebisch G (2009). "Regulation of potassium (K) handling in the renal collecting duct". Pflugers Arch. 458 (1): 157–68. doi:10.1007/s00424-008-0593-3. PMC 2730119. PMID 18839206.
↑ Giebisch GH, Wang WH (2010). "Potassium transport--an update". J Nephrol. 23 Suppl 16: S97–104. PMID 21170894.
↑ Magner PO, Robinson L, Halperin RM, Zettle R, Halperin ML (1988). "The plasma potassium concentration in metabolic acidosis: a re-evaluation". Am J Kidney Dis. 11 (3): 220–4. PMID 3344745.
↑ Giebisch G (1998). "Renal potassium transport: mechanisms and regulation". Am J Physiol. 274 (5 Pt 2): F817–33. PMID 9612319.

Template:WikiDoc Sources

[pmid18839206-1] Wang WH, Giebisch G (2009). "Regulation of potassium (K) handling in the renal collecting duct". Pflugers Arch. 458 (1): 157–68. doi:10.1007/s00424-008-0593-3. PMC 2730119. PMID 18839206.

[pmid21170894-2] Giebisch GH, Wang WH (2010). "Potassium transport--an update". J Nephrol. 23 Suppl 16: S97–104. PMID 21170894.

[pmid3344745-3] Magner PO, Robinson L, Halperin RM, Zettle R, Halperin ML (1988). "The plasma potassium concentration in metabolic acidosis: a re-evaluation". Am J Kidney Dis. 11 (3): 220–4. PMID 3344745.

[pmid9612319-4] Giebisch G (1998). "Renal potassium transport: mechanisms and regulation". Am J Physiol. 274 (5 Pt 2): F817–33. PMID 9612319.

[1]

[2]

[3]

[4]

@@ Line 1: / Line 1: @@
+__NOTOC__
+{| class="infobox" style="float:right;"
+|-
+| <figure-inline>[[image:Siren.gif|link=hyperkalemia resident survival guide|41x41px]]</figure-inline>|| <br> || <br>
+| [[Hyperkalemia resident survival guide|Resident <br> Survival   <br> Guide]]
+|}
+{{Hyperkalemia}}
+{{CMG}}; '''Associate Editor(s)-In-Chief:''' [[Priyamvada Singh|Priyamvada Singh, M.B.B.S.]] [mailto:psingh13579@gmail.com],[[Jogeet Singh Sekhon]]
+==Overview==
+The [[kidneys]] normally remove excess potassium from the body. Most cases of [[hyperkalemia]] occur in disorders that reduce the kidneys' ability to get rid of potassium. This may result from disorders such as [[acute kidney failure]], [[chronic kidney failure]] and [[glomerulonephritis]].
+==Risk Factors==
+* The [[kidneys]] normally remove excess [[potassium]] from the body
+* Most cases of [[hyperkalemia]] are due to  [[disorders]] that reduce the [[kidneys]]' ability to get rid of [[potassium]] <ref name="pmid18839206">{{cite journal| author=Wang WH, Giebisch G| title=Regulation of potassium (K) handling in the renal collecting duct. | journal=Pflugers Arch | year= 2009 | volume= 458 | issue= 1 | pages= 157-68 | pmid=18839206 | doi=10.1007/s00424-008-0593-3 | pmc=2730119 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=18839206  }} </ref><ref name="pmid21170894">{{cite journal| author=Giebisch GH, Wang WH| title=Potassium transport--an update. | journal=J Nephrol | year= 2010 | volume= 23 Suppl 16 | issue=  | pages= S97-104 | pmid=21170894 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=21170894  }} </ref>
+===Common risk factors===
+* The most common risk factors for hyperkalmeia include<ref name="pmid3344745">{{cite journal| author=Magner PO, Robinson L, Halperin RM, Zettle R, Halperin ML| title=The plasma potassium concentration in metabolic acidosis: a re-evaluation. | journal=Am J Kidney Dis | year= 1988 | volume= 11 | issue= 3 | pages= 220-4 | pmid=3344745 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=3344745  }} </ref>:
+**[[Acute kidney failure]]
+** [[Chronic kidney failure]]<ref name="pmid9612319">{{cite journal| author=Giebisch G| title=Renal potassium transport: mechanisms and regulation. | journal=Am J Physiol | year= 1998 | volume= 274 | issue= 5 Pt 2 | pages= F817-33 | pmid=9612319 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=9612319  }} </ref>
+** [[Diabetes mellitus]]
+** [[Glomerulonephritis]]
+** [[Obstructive uropathy]]
+** Rejection of a [[kidney transplant]]
+** [[Addison's disease]]
+* [[Burns]]
+* Disorders that cause [[blood cells]] to burst ([[hemolytic]] conditions)
+*[[Gastrointestinal]] [[bleeding]]
+* [[Rhabdomyolysis]] from drugs, alcoholism, coma, or certain infections
+*[[Surgery]]
+*[[Traumatic injury|Traumatic]] injury
+*[[Tumors]]
+* [[Acidosis]]
+* Medications
+**[[spironolactone]]
+** [[amiloride]]
+** [[triamterene]]
+**[[Potassium]] supplements (especially intravenous potassium).
+=== Less common risk factors ===
+* Fasting
+*[[Exercise]]
+*[[Fluoride]] [[toxicity]]
+*[[Hypoparathyroidism]]
+==References==
+{{reflist|2}}
+[[Category:Potassium]]
+[[Category:Medical emergencies]]
+[[Category:Endocrinology]]
+[[Category:Nephrology]]
+[[Category:Electrolyte disturbance]]
+[[Category:Blood tests]]
+[[Category:Emergency medicine]]
+[[Category:Intensive care medicine]]
+[[Category:Needs overview]]
+{{WikiDoc Help Menu}}
+{{WikiDoc Sources}}