Sandbox: jss: Difference between revisions

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{| class="wikitable" style="text-align:center"
{| border="1"
|+Multiplication table
|+ The table's caption
! Differentiating features !! Leukemia !! Leukemoid reaction
|-
|-
! Disease !! IgM levels !! IgG levels !! IgA levels !! IgE levels !! B cell defect !! T cell defect
! Causes
| Carcinogens || Infectious agents, biologically active substances and
products of tissue destruction
|-
|-
! IgM deficiency
! Pathogenesis
| ↓ || - || - || - || - || -
| The transformation of normal haematopoietic
cells to a tumor
| Activation of normal hematopoiesis and exit of immature leukocytes
into the bloodstream. 
|-
|-
! IgA deficiency
! Duration
| - || - || ↓ || - || - || -
| Chronic
| Temporary and reversible
|-
|-
! IgG deficiency
! Genetic level
| - || ↓ || - || - || - || -
| Defect
| No defect
|-
|-
! IgE deficiency
! Splenomegaly
| - || - || - || ↓ || - || -
| Present
| Not present
|-
|-
! Hypoproteinemia/Proteinuria
! Peripheral blood
| ↓ || ↓ || ↓ || ↓ || - || -
| Immature cells, pancytopenia
| Mature and immature granulocytes with left shift
|-
|-
! Comined Immunodeficiency
! Bone marrow
| ↓ || ↓ || ↓ || ↓ || + || +
| Increase in blasts and immature cells
| Myeloid hyperplasia and normal morphology
|-
|-
! X linked agammaglobulinemia
! LAP score
| ↓ || ↓ || ↓ || ↓ || + || -
| Low
| High
|-
|-
! Hyperimmunoglobulin M syndrome
! Toxic granules
| ↑ || ↓ || ↓ || ↓ || + || -
(suggestive of infection)
| Absent
| Present
|-
|-
! Common variable immunodeficiency
! Dohle inclusion bodies
| ↓ || ↓ || ↓ || ↓ || + || -
| Absent
|-
| Present
! Wiskott-Aldrich syndrome
| ↓ || ↓ || ↑ || ↑ || - || +
|-
|}

Latest revision as of 14:25, 9 October 2018


Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]


The table's caption
Differentiating features Leukemia Leukemoid reaction
Causes Carcinogens Infectious agents, biologically active substances and

products of tissue destruction

Pathogenesis The transformation of normal haematopoietic

cells to a tumor

Activation of normal hematopoiesis and exit of immature leukocytes

into the bloodstream.

Duration Chronic Temporary and reversible
Genetic level Defect No defect
Splenomegaly Present Not present
Peripheral blood Immature cells, pancytopenia Mature and immature granulocytes with left shift
Bone marrow Increase in blasts and immature cells Myeloid hyperplasia and normal morphology
LAP score Low High
Toxic granules 
(suggestive of infection)
Absent Present
Dohle inclusion bodies Absent Present
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