Constrictive pericarditis laboratory findings: Difference between revisions
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__NOTOC__ | __NOTOC__ | ||
{{Constrictive pericarditis}} | {{Constrictive pericarditis}} | ||
{{CMG}} | {{CMG}}; {{AE}} {{Hudakarman}} | ||
==Overview== | ==Overview== | ||
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*Proteinuria (nephrotic range) | *Proteinuria (nephrotic range) | ||
*Elevated sedimentation rate (ESR), C-reactive protein (CRP) level (postpericardiotomy syndrome) | *Elevated sedimentation rate (ESR), C-reactive protein (CRP) level (postpericardiotomy syndrome) | ||
*Brain natriuretic peptide (BNP), mildly increased (<150 ng/L). | *Brain natriuretic peptide (BNP), mildly increased (<150 ng/L)<ref name="pmid159366242">{{cite journal| author=Leya FS, Arab D, Joyal D, Shioura KM, Lewis BE, Steen LH et al.| title=The efficacy of brain natriuretic peptide levels in differentiating constrictive pericarditis from restrictive cardiomyopathy. | journal=J Am Coll Cardiol | year= 2005 | volume= 45 | issue= 11 | pages= 1900-2 | pmid=15936624 | doi=10.1016/j.jacc.2005.03.050 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=15936624 }}</ref> | ||
*Antinuclear antibody (ANA) or rheumatoid factor (RF) (associated collagen vascular disorder) | *Antinuclear antibody (ANA) or rheumatoid factor (RF) (associated collagen vascular disorder) | ||
*Purified protein derivative (PPD) skin test (TB) | *Purified protein derivative (PPD) skin test (TB) | ||
Latest revision as of 18:25, 27 December 2019
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Constrictive Pericarditis Microchapters |
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Differentiating Constrictive Pericarditis from other Diseases |
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Treatment |
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Case Studies |
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Constrictive pericarditis laboratory findings On the Web |
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American Roentgen Ray Society Images of Constrictive pericarditis laboratory findings |
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Risk calculators and risk factors for Constrictive pericarditis laboratory findings |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Huda A. Karman, M.D.
Overview
Constrictive pericarditis lab findings can show evidence of congestive heart failure, or associated protein losing enteropathy, nephrotic syndrome, and LFT abnormalities consistent with hepatic congestion and chylous ascites
Laboratory findings
- Complete blood count (CBC)
- Dilutional anemia if congestive heart failure (CHF) is present
- Normocytic normochromic anemia (chronic inflammation)
- Leukocytosis
- Infectious, bacterial, or rheumatologic etiology source or steroid therapy
- Leukopenia
- Malignancy and chemotherapeutic agents use
- Hyponatremia or pseudohyponatremia
- Contraction alkalosis (hypochloremia with hypercarbia)
- Elevation of blood urea nitrogen (BUN) levels f
- Serum creatinine levels are observed
- Arterial blood gas measurement
- Metabolic acidosis (ie, low pH and low bicarbonate),
- Compensatory respiratory alkalosis (ie, decreased partial pressure of carbon dioxide
- Elevated transaminase levels
- Hypoalbuminemia (a protein-losing enteropathy (PLE)
- Proteinuria (nephrotic range)
- Elevated sedimentation rate (ESR), C-reactive protein (CRP) level (postpericardiotomy syndrome)
- Brain natriuretic peptide (BNP), mildly increased (<150 ng/L)[1]
- Antinuclear antibody (ANA) or rheumatoid factor (RF) (associated collagen vascular disorder)
- Purified protein derivative (PPD) skin test (TB)
References
- ↑ Leya FS, Arab D, Joyal D, Shioura KM, Lewis BE, Steen LH; et al. (2005). "The efficacy of brain natriuretic peptide levels in differentiating constrictive pericarditis from restrictive cardiomyopathy". J Am Coll Cardiol. 45 (11): 1900–2. doi:10.1016/j.jacc.2005.03.050. PMID 15936624.