Tricuspid atresia laboratory findings: Difference between revisions
Line 5: | Line 5: | ||
{{CMG}} '''Associate Editor-In-Chief:'''{{Sara.Zand}} | {{CMG}} '''Associate Editor-In-Chief:'''{{Sara.Zand}} | ||
==Overview== | ==Overview== | ||
In [[cyanotic]] older patients laboratory | In [[cyanotic]] older patients laboratory findings may include:[[Polycythemia]] due to secondary [[erythrocytosis]], elevated [[prothrombin time]] and [[partial thromboplastin time]], decreased levels of factors 5,7,8,9: qualitative and quantitative, [[platelet disorder]], increased [[fibrinolysis]] and paradoxical [[thrombotic]] tendency, [[proteinuria]], [[hyperuricemia]], [[renal failure]], [[uric acid]] [[nephrolithiasis]] | ||
==[[Tricuspid atresia]] laboratory findings== | ==[[Tricuspid atresia]] laboratory findings== |
Latest revision as of 16:57, 8 November 2020
Tricuspid atresia Microchapters |
Diagnosis |
---|
Treatment |
Special Scenarios |
Case Studies |
Tricuspid atresia laboratory findings On the Web |
American Roentgen Ray Society Images of Tricuspid atresia laboratory findings |
Risk calculators and risk factors for Tricuspid atresia laboratory findings |
Tricuspid atresia Microchapters |
Diagnosis |
---|
Treatment |
Special Scenarios |
Case Studies |
Tricuspid atresia laboratory findings On the Web |
American Roentgen Ray Society Images of Tricuspid atresia laboratory findings |
Risk calculators and risk factors for Tricuspid atresia laboratory findings |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor-In-Chief:Sara Zand, M.D.[2]
Overview
In cyanotic older patients laboratory findings may include:Polycythemia due to secondary erythrocytosis, elevated prothrombin time and partial thromboplastin time, decreased levels of factors 5,7,8,9: qualitative and quantitative, platelet disorder, increased fibrinolysis and paradoxical thrombotic tendency, proteinuria, hyperuricemia, renal failure, uric acid nephrolithiasis
Tricuspid atresia laboratory findings
In cyanotic older patients laboratory finding may include:[1]
- Polycythemia due to secondary erythrocytosis
- Elevated prothrombin time and partial thromboplastin time
- Decreased levels of factors 5,7,8,9: qualitative and quantitative
- Platelet disorder
- Increased fibrinolysis and paradoxical thrombotic tendency
- Proteinuria
- Hyperuricemia
- Renal failure
- Uric acid nephrolithiasis
References
- ↑ Bhardwaj V, Malhotra P, Hasija S, Chowdury UK, Pangasa N (2017). "Coagulopathies in cyanotic cardiac patients: An analysis with three point - of - care testing devices (Thromboelastography, rotational thromboelastometry, and sonoclot analyzer)". Ann Card Anaesth. 20 (2): 212–218. doi:10.4103/aca.ACA_4_17. PMC 5408528. PMID 28393783.