Alcohol withdrawal other diagnostic studies: Difference between revisions
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==Overview== | ==Overview== | ||
There are no other [[diagnostic studies]] associated with [[alcohol]] withdrawal. However, studies with [[neurochemical]] brain [[imaging]] have shown the [[neurochemical]] | There are no other [[diagnostic studies]] associated with [[alcohol]] withdrawal. However, studies with [[neurochemical]] brain [[imaging]] have shown the [[neurochemical]] effects during [[alcohol]] dependence and withdrawal. | ||
==Other Diagnostic Studies== | ==Other Diagnostic Studies== | ||
* There are no other [[diagnostic studies]] associated with [[alcohol]] withdrawal. | * There are no other [[diagnostic studies]] associated with [[alcohol]] withdrawal. | ||
* However, studies with [[neurochemical]] brain [[imaging]] [with magnetic resonance spectrometry (MRS), positron emission tomography (PET), and single photon emission computed tomography (SPECT)] have shown the [[neurochemical]] ([[GABA]], [[glutamate]], and [[dopamine]]) effects during [[alcohol]] dependence and withdrawal such as:<ref name="pmid26510169">{{cite journal| author=Hillmer AT, Mason GF, Fucito LM, O'Malley SS, Cosgrove KP| title=How Imaging Glutamate, γ-Aminobutyric Acid, and Dopamine Can Inform the Clinical Treatment of Alcohol Dependence and Withdrawal. | journal=Alcohol Clin Exp Res | year= 2015 | volume= 39 | issue= 12 | pages= 2268-82 | pmid=26510169 | doi=10.1111/acer.12893 | pmc=4712074 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=26510169 }} </ref> | * However, studies with [[neurochemical]] brain [[imaging]] [with [[MRS|magnetic resonance spectrometry (MRS)]], [[PET|positron emission tomography (PET)]], and [[SPECT|single photon emission computed tomography (SPECT)]]] have shown the [[neurochemical]] ([[GABA]], [[glutamate]], and [[dopamine]]) effects during [[alcohol]] dependence and withdrawal such as:<ref name="pmid26510169">{{cite journal| author=Hillmer AT, Mason GF, Fucito LM, O'Malley SS, Cosgrove KP| title=How Imaging Glutamate, γ-Aminobutyric Acid, and Dopamine Can Inform the Clinical Treatment of Alcohol Dependence and Withdrawal. | journal=Alcohol Clin Exp Res | year= 2015 | volume= 39 | issue= 12 | pages= 2268-82 | pmid=26510169 | doi=10.1111/acer.12893 | pmc=4712074 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=26510169 }} </ref> | ||
** [[Downregulation]] of [[dopamine]] D2/D3 receptor | ** [[Downregulation]] of [[dopamine]] D2/D3 receptor | ||
** Higher [[glutamate]] levels during acute [[alcohol]] withdrawal followed by fluctuating concentrations | ** Higher [[glutamate]] levels during acute [[alcohol]] withdrawal followed by fluctuating concentrations | ||
Latest revision as of 13:54, 13 November 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Shakiba Hassanzadeh, MD[2]
Overview
There are no other diagnostic studies associated with alcohol withdrawal. However, studies with neurochemical brain imaging have shown the neurochemical effects during alcohol dependence and withdrawal.
Other Diagnostic Studies
- There are no other diagnostic studies associated with alcohol withdrawal.
- However, studies with neurochemical brain imaging [with magnetic resonance spectrometry (MRS), positron emission tomography (PET), and single photon emission computed tomography (SPECT)] have shown the neurochemical (GABA, glutamate, and dopamine) effects during alcohol dependence and withdrawal such as:[1]
References
- ↑ Hillmer AT, Mason GF, Fucito LM, O'Malley SS, Cosgrove KP (2015). "How Imaging Glutamate, γ-Aminobutyric Acid, and Dopamine Can Inform the Clinical Treatment of Alcohol Dependence and Withdrawal". Alcohol Clin Exp Res. 39 (12): 2268–82. doi:10.1111/acer.12893. PMC 4712074. PMID 26510169.