Granulomatous amoebic encephalitis causes: Difference between revisions
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==Overview== | ==Overview== | ||
Balamuthia mandrillaris has only recently been isolated from the environment and has also been isolated from autopsy specimens of infected humans and animals. B. mandrillaris has only two stages, [[cyst]]s and [[trophozoites]] , in its life cycle. No flagellated stage exists as part of the life cycle. The [[trophozoites]] replicate by [[mitosis]] (nuclear membrane does not remain intact) . The trophozoites are the infective forms, although both [[cyst]]s and [[trophozoites]] gain entry into the body through various means. Entry can occur through the nasal passages to the lower respiratory tract , or ulcerated or broken skin . When B. mandrillaris enters the respiratory system or through the [[skin]], it can invade the [[central nervous system]] by hematogenous dissemination causing [[granulomatous amebic encephalitis]] (GAE) or disseminated disease , or skin lesions in individuals who are immune competent as well as those with compromised immune systems. B. mandrillaris [[cyst]]s and [[trophozoites]] are found in tissue. | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
[[Category:Neurology]] | [[Category:Neurology]] | ||
{{WikiDoc Help Menu}} | {{WikiDoc Help Menu}} | ||
{{wikiDoc Sources}} | {{wikiDoc Sources}} |
Latest revision as of 17:50, 18 September 2017
Granulomatous amoebic encephalitis Microchapters |
Differentiating Granulomatous amoebic encephalitis from other Diseases |
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Diagnosis |
Treatment |
Case Studies |
Granulomatous amoebic encephalitis causes On the Web |
American Roentgen Ray Society Images of Granulomatous amoebic encephalitis causes |
Directions to Hospitals Treating Granulomatous amoebic encephalitis |
Risk calculators and risk factors for Granulomatous amoebic encephalitis causes |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Balamuthia mandrillaris has only recently been isolated from the environment and has also been isolated from autopsy specimens of infected humans and animals. B. mandrillaris has only two stages, cysts and trophozoites , in its life cycle. No flagellated stage exists as part of the life cycle. The trophozoites replicate by mitosis (nuclear membrane does not remain intact) . The trophozoites are the infective forms, although both cysts and trophozoites gain entry into the body through various means. Entry can occur through the nasal passages to the lower respiratory tract , or ulcerated or broken skin . When B. mandrillaris enters the respiratory system or through the skin, it can invade the central nervous system by hematogenous dissemination causing granulomatous amebic encephalitis (GAE) or disseminated disease , or skin lesions in individuals who are immune competent as well as those with compromised immune systems. B. mandrillaris cysts and trophozoites are found in tissue.
References