Encephalopathy future or investigational therapies: Difference between revisions
Jump to navigation
Jump to search
No edit summary |
|||
(One intermediate revision by one other user not shown) | |||
Line 3: | Line 3: | ||
==Overview== | ==Overview== | ||
Newer investigational and therapeutic modalities are | Newer investigational and therapeutic modalities are under consideration. | ||
==New Therapies== | ==New Therapies== | ||
Newer modalities of treatment are considered for [[hypoxic encephalopathy]]. These drugs reduce the brain activity and its oxygen | Newer modalities of treatment are considered for [[hypoxic encephalopathy]]. These drugs reduce the brain activity and its oxygen requirements. | ||
* Magnesium - blocks NMDA receptor | * Magnesium - blocks NMDA receptor | ||
* [[Adenosine]] - decreases glutamate release | * [[Adenosine]] - decreases glutamate release |
Latest revision as of 12:30, 24 July 2012
Encephalopathy |
Diagnosis |
---|
Treatment |
Encephalopathy future or investigational therapies On the Web |
American Roentgen Ray Society Images of Encephalopathy future or investigational therapies |
Encephalopathy future or investigational therapies in the news |
Risk calculators and risk factors for Encephalopathy future or investigational therapies |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Aditya Govindavarjhulla, M.B.B.S. [2]
Overview
Newer investigational and therapeutic modalities are under consideration.
New Therapies
Newer modalities of treatment are considered for hypoxic encephalopathy. These drugs reduce the brain activity and its oxygen requirements.
- Magnesium - blocks NMDA receptor
- Adenosine - decreases glutamate release
- Vitamin C - reduces free radicals
- Caspase inhibitors - Inhibit apoptosis and prevents death of brain tissue