Calciphylaxis pathophysiology: Difference between revisions
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==Overview== | |||
==Pathophysiology== | |||
Calciphylaxis is characterised by | |||
# systemic medial calcification of the arteries, i.e. calcification of [[tunica media]]. Unlike other forms of vascular calcifications (eg, intimal, medial, valvular), calciphylaxis is characterised also by | |||
# small vessel mural calcification with or without endovascular fibrosis, extravascular calcification and vascular thrombosis, leading to tissue ischaemia (including skin ischaemia and, hence, skin [[necrosis]]). | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
[[Category:Disease]] | [[Category:Disease]] | ||
Latest revision as of 15:35, 28 September 2012
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Calciphylaxis Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Calciphylaxis pathophysiology On the Web |
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American Roentgen Ray Society Images of Calciphylaxis pathophysiology |
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Risk calculators and risk factors for Calciphylaxis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
Calciphylaxis is characterised by
- systemic medial calcification of the arteries, i.e. calcification of tunica media. Unlike other forms of vascular calcifications (eg, intimal, medial, valvular), calciphylaxis is characterised also by
- small vessel mural calcification with or without endovascular fibrosis, extravascular calcification and vascular thrombosis, leading to tissue ischaemia (including skin ischaemia and, hence, skin necrosis).