Dysentery natural history, complications and prognosis: Difference between revisions
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Please help WikiDoc by adding more content here. It's easy! Click [[Help:How_to_Edit_a_Page|here]] to learn about editing. | Please help WikiDoc by adding more content here. It's easy! Click [[Help:How_to_Edit_a_Page|here]] to learn about editing. | ||
{{CMG}} | {{CMG}} {{AE}} {{KD}} | ||
==Natural History== | ==Natural History== | ||
Persons with diarrhea usually recover completely, although it may be several months before their bowel habits are entirely normal. | Persons with diarrhea usually recover completely, although it may be several months before their bowel habits are entirely normal. | ||
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* [[Shock]] | * [[Shock]] | ||
* [[Delirium]] | * [[Delirium]] | ||
*[[Hemolytic uremic syndrome]]: EHECs and shigella that induce bloody diarrhea can lead to HUS. The clinical manifestations of postdiarrheal HUS include [[acute kidney injury|acute renal failure]], [[microangiopathic hemolytic anemia]], and [[thrombocytopenia]]. The verocytotoxin (shiga-like toxin) can directly damage renal and endothelial cells. Thrombocytopenia occurs as platelets are consumed by clotting. Hemolytic anemia results from intravascular [[fibrin]] deposition, increased fragility of red blood cells, and fragmentation. | *[[Hemolytic uremic syndrome]]: EHECs and shigella that induce bloody diarrhea can lead to HUS. The clinical manifestations of postdiarrheal HUS include [[acute kidney injury|acute renal failure]], [[microangiopathic hemolytic anemia]], and [[thrombocytopenia]]. The verocytotoxin (shiga-like toxin) can directly damage renal and endothelial cells. Thrombocytopenia occurs as platelets are consumed by clotting. Hemolytic anemia results from intravascular [[fibrin]] deposition, increased fragility of red blood cells, and fragmentation. | ||
* [[Thrombotic thrombocytopenic purpura]] | * [[Thrombotic thrombocytopenic purpura]] | ||
* [[Reactive arthritis]] | * [[Reactive arthritis]] | ||
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{{Reflist|2}} | {{Reflist|2}} | ||
[[Category:Needs content]] | [[Category:Needs content]] | ||
[[Category:Water-borne diseases]] | [[Category:Water-borne diseases]] | ||
[[Category:Conditions diagnosed by stool test]] | [[Category:Conditions diagnosed by stool test]] | ||
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Latest revision as of 17:37, 18 September 2017
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Kalsang Dolma, M.B.B.S.[2]
Natural History
Persons with diarrhea usually recover completely, although it may be several months before their bowel habits are entirely normal.
Complications
- Shock
- Delirium
- Hemolytic uremic syndrome: EHECs and shigella that induce bloody diarrhea can lead to HUS. The clinical manifestations of postdiarrheal HUS include acute renal failure, microangiopathic hemolytic anemia, and thrombocytopenia. The verocytotoxin (shiga-like toxin) can directly damage renal and endothelial cells. Thrombocytopenia occurs as platelets are consumed by clotting. Hemolytic anemia results from intravascular fibrin deposition, increased fragility of red blood cells, and fragmentation.
- Thrombotic thrombocytopenic purpura
- Reactive arthritis
Prognosis
With correct treatment, most cases of amoebic and bacterial dysentery subside within ten days, and most individuals will achieve a full recovery within two to four weeks after beginning proper treatment. If the disease is left untreated, the prognosis varies with the immune status of the individual patient and the severity of disease. Extreme dehydration can prolong recovery and significantly raises the risk for serious complications.[1]
References
- ↑ mdguidelines.com. "Dysentery-Prognosis". Retrieved 2010-11-17.