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==Puerperal Mastitis==
==Overview==


Caused by the blocking of the [[Lactiferous duct|milk duct]]s while the mother is lactating (see [[breastfeeding]]). It can cause painful areas on the breasts or [[nipple]]s and may lead to a [[fever]] or [[influenza|flu]]-like symptoms. Except in heavy cases it is not necessary to wean a [[infant|nursling]] because of mastitis; in fact, nursing is the most effective way to remove the blockage and alleviate the symptoms, and is not harmful to the baby. Sudden weaning can cause or exacerbate mastitis symptoms.
Mastitis can be classified according to several subtypes based on the [[etiology]], duration of the [[disease]], [[anatomical]] location, [[immunological]] association and age of the [[patient]].


Mastitis can be discerned from simple blockages by the intensity of pain, heat emanating from the area, redness and fever in the mother. In some cases the fever can become severe, requiring [[antibiotic]]s; ten percent of cases develop into [[abscess]]es that need to be drained surgically.
==Classification==


==Nonpuerperal Mastitis==
Mastitis can be classified according to several subtypes based on the [[etiology]], duration of the [[disease]], [[anatomical]] location, [[immunological]] association and age of the [[patient]] as follows:


The term Nonpuerperal mastitis describes all inflammatory lesions of the breast except inflammatory breast cancer and skin related conditions like dermatitis and foliculitis. This article includes description of mastitis as well as various kinds of mammary abscesses.
===Puerperal vs. Non-puerperal Mastitis===


Most patients are women of reproductive age but mastitis beyond the age of 60 is not uncommon. Rarely occurs in newborn children (Mastitis neonatorum) or prepubertal children.  
This classification is important when counseling patients on prevention because improper latch of the baby to the breast may result in [[nipple]] [[injuries]].


So called pre-pubertal mastitis can occur shortly before or during the first stages of puberty of both boys and girls with very mild symptoms and resolves without intervention.
===Chronic vs. Acute Mastitis===
No specific timeline has been used to define the chronicity of the [[infection]].
 
===Periductal vs. Ductal===
This classification is based on the [[anatomical]] location of the [[inflammatory]] process and may not be clinically important.
 
===Autoimmune vs. Non-autoimmune Mastitis===
 
Examples of [[autoimmune]] mastitis include [[granulomatous]] and [[lupus]] mastitis.  This classification is especially important because [[granulomatous]] and [[lupus]] mastitis are treated differently from other types of mastitis.<ref name="pmid26148520">{{cite journal| author=Altintoprak F, Kivilcim T, Yalkin O, Uzunoglu Y, Kahyaoglu Z, Dilek ON| title=Topical Steroids Are Effective in the Treatment of Idiopathic Granulomatous Mastitis. | journal=World J Surg | year= 2015 | volume= 39 | issue= 11 | pages= 2718-23 | pmid=26148520 | doi=10.1007/s00268-015-3147-9 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=26148520}}</ref><ref name="pmid20030652">{{cite journal| author=Ocal K, Dag A, Turkmenoglu O, Kara T, Seyit H, Konca K| title=Granulomatous mastitis: clinical, pathological features, and management. | journal=Breast J | year= 2010 | volume= 16 | issue= 2 | pages= 176-82 | pmid=20030652 | doi=10.1111/j.1524-4741.2009.00879.x | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=20030652}}</ref><ref name="pmid19098467">{{cite journal| author=Summers TA, Lehman MB, Barner R, Royer MC| title=Lupus mastitis: a clinicopathologic review and addition of a case. | journal=Adv Anat Pathol | year= 2009 | volume= 16 | issue= 1 | pages= 56-61 | pmid=19098467 | doi=10.1097/PAP.0b013e3181915ff7 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=19098467}}</ref>
 
===Pre-pubertal Mastitis===
 
This is usullay [[self-limiting]] and may not need any [[antibiotic]] therapy.


==References==
==References==
{{reflist|2}}
{{reflist|2}}


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Latest revision as of 22:38, 29 July 2020

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Prince Tano Djan, BSc, MBChB [2]

Overview

Mastitis can be classified according to several subtypes based on the etiology, duration of the disease, anatomical location, immunological association and age of the patient.

Classification

Mastitis can be classified according to several subtypes based on the etiology, duration of the disease, anatomical location, immunological association and age of the patient as follows:

Puerperal vs. Non-puerperal Mastitis

This classification is important when counseling patients on prevention because improper latch of the baby to the breast may result in nipple injuries.

Chronic vs. Acute Mastitis

No specific timeline has been used to define the chronicity of the infection.

Periductal vs. Ductal

This classification is based on the anatomical location of the inflammatory process and may not be clinically important.

Autoimmune vs. Non-autoimmune Mastitis

Examples of autoimmune mastitis include granulomatous and lupus mastitis. This classification is especially important because granulomatous and lupus mastitis are treated differently from other types of mastitis.[1][2][3]

Pre-pubertal Mastitis

This is usullay self-limiting and may not need any antibiotic therapy.

References

  1. Altintoprak F, Kivilcim T, Yalkin O, Uzunoglu Y, Kahyaoglu Z, Dilek ON (2015). "Topical Steroids Are Effective in the Treatment of Idiopathic Granulomatous Mastitis". World J Surg. 39 (11): 2718–23. doi:10.1007/s00268-015-3147-9. PMID 26148520.
  2. Ocal K, Dag A, Turkmenoglu O, Kara T, Seyit H, Konca K (2010). "Granulomatous mastitis: clinical, pathological features, and management". Breast J. 16 (2): 176–82. doi:10.1111/j.1524-4741.2009.00879.x. PMID 20030652.
  3. Summers TA, Lehman MB, Barner R, Royer MC (2009). "Lupus mastitis: a clinicopathologic review and addition of a case". Adv Anat Pathol. 16 (1): 56–61. doi:10.1097/PAP.0b013e3181915ff7. PMID 19098467.

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