Brugada syndrome physical examination: Difference between revisions
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{{Brugada syndrome}} | {{Brugada syndrome}} | ||
{{CMG}} | {{CMG}} {{AE}} {{S.G.}} | ||
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==Overview== | ==Overview== | ||
[[Patients]] with Brugada syndrome usually appear usually normal. [[Physical examination]] of [[patients]] with Brugada syndrome is usually remarkable by [[vagal maneuvers]] such as [[carotid sinus massage]] may increase [[vagal]] tone and may unmask the presence of a Type I Brugada pattern. | |||
The presence of recent [[symptoms]] such as [[syncope]], it is important to check the temperature in so far as [[fever]] may trigger a self terminating or sustained episode of [[ventricular tachycardia]] / [[ventricular fibrillation]]. The presence of [[fever]] is also a target of [[Antipyretic|antipyretic therap]]<nowiki/>y on [[physical examination]] is highly suggestive of Brugada syndrome. | |||
==Physical Examination== | |||
*[[Patients]] with Brugada syndrome usually appear usually normal. [[Physical examination]] of [[patients]] with Brugada syndrome is usually remarkable by [[vagal maneuvers]] such as [[carotid sinus massage]] may increase [[vagal]] tone and may unmask the presence of a Type I Brugada pattern.<ref name="CrossonHanash20102">{{cite journal|last1=Crosson|first1=JaneE|last2=Hanash|first2=CarlaR|title=Emergency diagnosis and management of pediatric arrhythmias|journal=Journal of Emergencies, Trauma, and Shock|volume=3|issue=3|year=2010|pages=251|issn=0974-2700|doi=10.4103/0974-2700.66525}}</ref> | |||
*<nowiki/>The presence of recent [[symptoms]] such as [[syncope]], it is important to check the temperature in so far as [[fever]] may trigger a self terminating or sustained episode of [[ventricular tachycardia]] / [[ventricular fibrillation]]. The presence of [[fever]] is also a target of [[Antipyretic|antipyretic therap]]<nowiki/>y on [[physical examination]] is highly suggestive of Brugada syndrome.<ref name="Al-KhatibStevenson2018">{{cite journal|last1=Al-Khatib|first1=Sana M.|last2=Stevenson|first2=William G.|last3=Ackerman|first3=Michael J.|last4=Bryant|first4=William J.|last5=Callans|first5=David J.|last6=Curtis|first6=Anne B.|last7=Deal|first7=Barbara J.|last8=Dickfeld|first8=Timm|last9=Field|first9=Michael E.|last10=Fonarow|first10=Gregg C.|last11=Gillis|first11=Anne M.|last12=Granger|first12=Christopher B.|last13=Hammill|first13=Stephen C.|last14=Hlatky|first14=Mark A.|last15=Joglar|first15=José A.|last16=Kay|first16=G. Neal|last17=Matlock|first17=Daniel D.|last18=Myerburg|first18=Robert J.|last19=Page|first19=Richard L.|title=2017 AHA/ACC/HRS Guideline for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death|journal=Circulation|volume=138|issue=13|year=2018|issn=0009-7322|doi=10.1161/CIR.0000000000000549}}</ref> | |||
=== Heart === | |||
*[[Cardiovascular]] [[Physical examination|examination]] is required to rule out other possible [[Sudden cardiac death|cardiac]] causes of [[syncope]].<ref name="Arnar2013">{{cite journal|last1=Arnar|first1=David O.|title=Syncope in patients with structural heart disease|journal=Journal of Internal Medicine|volume=273|issue=4|year=2013|pages=336–344|issn=09546820|doi=10.1111/joim.12027}}</ref> | |||
**[[Heart murmur|Heart murmurs]] from [[Hypertrophy (medical)|hypertrophic]] [[cardiomyopathy]] | |||
**[[Valvular heart disease|Valvular]] or [[septal defect]] | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
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{{WS}} | |||
[[CME Category::Cardiology|CME Category::]]<nowiki/>[[CME Category::Cardiology|Cardiology]] | |||
[[Category:Needs content]] | [[Category:Needs content]] | ||
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[[Category:Channelopathy]] | [[Category:Channelopathy]] | ||
[[Category:Genetic disorders]] | [[Category:Genetic disorders]] | ||
Latest revision as of 16:32, 13 January 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Sogand Goudarzi, MD [2]
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Overview
Patients with Brugada syndrome usually appear usually normal. Physical examination of patients with Brugada syndrome is usually remarkable by vagal maneuvers such as carotid sinus massage may increase vagal tone and may unmask the presence of a Type I Brugada pattern.
The presence of recent symptoms such as syncope, it is important to check the temperature in so far as fever may trigger a self terminating or sustained episode of ventricular tachycardia / ventricular fibrillation. The presence of fever is also a target of antipyretic therapy on physical examination is highly suggestive of Brugada syndrome.
Physical Examination
- Patients with Brugada syndrome usually appear usually normal. Physical examination of patients with Brugada syndrome is usually remarkable by vagal maneuvers such as carotid sinus massage may increase vagal tone and may unmask the presence of a Type I Brugada pattern.[1]
- The presence of recent symptoms such as syncope, it is important to check the temperature in so far as fever may trigger a self terminating or sustained episode of ventricular tachycardia / ventricular fibrillation. The presence of fever is also a target of antipyretic therapy on physical examination is highly suggestive of Brugada syndrome.[2]
Heart
- Cardiovascular examination is required to rule out other possible cardiac causes of syncope.[3]
References
- ↑ Crosson, JaneE; Hanash, CarlaR (2010). "Emergency diagnosis and management of pediatric arrhythmias". Journal of Emergencies, Trauma, and Shock. 3 (3): 251. doi:10.4103/0974-2700.66525. ISSN 0974-2700.
- ↑ Al-Khatib, Sana M.; Stevenson, William G.; Ackerman, Michael J.; Bryant, William J.; Callans, David J.; Curtis, Anne B.; Deal, Barbara J.; Dickfeld, Timm; Field, Michael E.; Fonarow, Gregg C.; Gillis, Anne M.; Granger, Christopher B.; Hammill, Stephen C.; Hlatky, Mark A.; Joglar, José A.; Kay, G. Neal; Matlock, Daniel D.; Myerburg, Robert J.; Page, Richard L. (2018). "2017 AHA/ACC/HRS Guideline for Management of Patients With Ventricular Arrhythmias and the Prevention of Sudden Cardiac Death". Circulation. 138 (13). doi:10.1161/CIR.0000000000000549. ISSN 0009-7322.
- ↑ Arnar, David O. (2013). "Syncope in patients with structural heart disease". Journal of Internal Medicine. 273 (4): 336–344. doi:10.1111/joim.12027. ISSN 0954-6820.