Contrast induced nephropathy history and symptoms: Difference between revisions
No edit summary |
|||
| (One intermediate revision by the same user not shown) | |||
| Line 7: | Line 7: | ||
==History and Symptoms== | ==History and Symptoms== | ||
In most of cases, the increase in creatinine occurs within 24 to 48 hours of contrast administration, which reflect decline in [[GFR]], the creatinine usually starts to decline within three to seven days, other laboratory manifestations may be present, including [[hyperkalemia]], [[acidosis]] and [[hyperphosphatemia]].<ref name="pmid7942832">{{cite journal| author=Rudnick MR, Berns JS, Cohen RM, Goldfarb S| title=Nephrotoxic risks of renal angiography: contrast media-associated nephrotoxicity and atheroembolism--a critical review. | journal=Am J Kidney Dis | year= 1994 | volume= 24 | issue= 4 | pages= 713-27 | pmid=7942832 | doi= | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=7942832 }} </ref> Other symptoms of [[Acute kidney injury]] may be present. | In most of cases, the increase in creatinine occurs within 24 to 48 hours of contrast administration, which reflect decline in [[GFR]], the creatinine usually starts to decline within three to seven days, other laboratory manifestations may be present, including [[hyperkalemia]], [[acidosis]] and [[hyperphosphatemia]].<ref name="pmid7942832">{{cite journal| author=Rudnick MR, Berns JS, Cohen RM, Goldfarb S| title=Nephrotoxic risks of renal angiography: contrast media-associated nephrotoxicity and atheroembolism--a critical review. | journal=Am J Kidney Dis | year= 1994 | volume= 24 | issue= 4 | pages= 713-27 | pmid=7942832 | doi= | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=7942832 }} </ref> Other symptoms of [[Acute kidney injury]] may be present. | ||
==References== | ==References== | ||
Latest revision as of 17:57, 16 September 2013
|
Contrast Induced Nephropathy Microchapters |
|
Differentiating Contrast induced nephropathy from other Diseases |
|---|
|
Diagnosis |
|
Treatment |
|
Case Studies |
|
Contrast induced nephropathy history and symptoms On the Web |
|
American Roentgen Ray Society Images of Contrast induced nephropathy history and symptoms |
|
Contrast induced nephropathy history and symptoms in the news |
|
Directions to Hospitals Treating Contrast induced nephropathy |
|
Risk calculators and risk factors for Contrast induced nephropathy history and symptoms |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Mohamed Moubarak, M.D. [2]
Overview
Creatinine increase is the characteristic finding in CIN, kidney injury occure with in minutes of exposure to contrast agents, however clinical manifestations such as oliguria or elevation of serum creatinine are generally observed within 24 to 48 hours after contrast exposure.
History and Symptoms
In most of cases, the increase in creatinine occurs within 24 to 48 hours of contrast administration, which reflect decline in GFR, the creatinine usually starts to decline within three to seven days, other laboratory manifestations may be present, including hyperkalemia, acidosis and hyperphosphatemia.[1] Other symptoms of Acute kidney injury may be present.
References
- ↑ Rudnick MR, Berns JS, Cohen RM, Goldfarb S (1994). "Nephrotoxic risks of renal angiography: contrast media-associated nephrotoxicity and atheroembolism--a critical review". Am J Kidney Dis. 24 (4): 713–27. PMID 7942832.