Interstitial nephritis causes: Difference between revisions
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Common causes include infection, or reaction to medication (such as an [[analgesic]] or [[antibiotic]]s). 71%<ref name="pmid14671029">{{cite journal |author=Baker R, Pusey C |title=The changing profile of acute tubulointerstitial nephritis |journal=Nephrol Dial Transplant |volume=19 |issue=1 |pages=8-11 |year=2004 |pmid=14671029|url=http://ndt.oxfordjournals.org/cgi/content/full/19/1/8}}</ref> to 92%<ref name="pmid15340098">{{cite journal |author=Clarkson M, Giblin L, O'Connell F, O'Kelly P, Walshe J, Conlon P, O'Meara Y, Dormon A, Campbell E, Donohoe J |title=Acute interstitial nephritis: clinical features and response to corticosteroid therapy |journal=Nephrol Dial Transplant |volume=19 |issue=11 |pages=2778-83 |year=2004 |pmid=15340098}}</ref> of cases are reported to be caused by drugs. This disease is also caused by other diseases and toxins that do damage to the kidney. Both acute and chronic tubulointerstitial nephritis can be caused by a bacterial infection in the kidneys, known as [[pyelonephritis]]. The most common cause is by an allergic reaction to a drug. The drugs that are known to cause this sort of reaction are antibiotics such as penicillin, and nonsteroidal anti-inflammatory drugs, such as aspirin. The time between exposure to the drug and the development of acute tubulointerstitial nephritis can be anywhere from 5 days to 5 weeks. | Common causes include infection, or reaction to medication (such as an [[analgesic]] or [[antibiotic]]s). 71%<ref name="pmid14671029">{{cite journal |author=Baker R, Pusey C |title=The changing profile of acute tubulointerstitial nephritis |journal=Nephrol Dial Transplant |volume=19 |issue=1 |pages=8-11 |year=2004 |pmid=14671029|url=http://ndt.oxfordjournals.org/cgi/content/full/19/1/8}}</ref> to 92%<ref name="pmid15340098">{{cite journal |author=Clarkson M, Giblin L, O'Connell F, O'Kelly P, Walshe J, Conlon P, O'Meara Y, Dormon A, Campbell E, Donohoe J |title=Acute interstitial nephritis: clinical features and response to corticosteroid therapy |journal=Nephrol Dial Transplant |volume=19 |issue=11 |pages=2778-83 |year=2004 |pmid=15340098}}</ref> of cases are reported to be caused by drugs. This disease is also caused by other diseases and toxins that do damage to the kidney. Both acute and chronic tubulointerstitial nephritis can be caused by a bacterial infection in the kidneys, known as [[pyelonephritis]]. The most common cause is by an allergic reaction to a drug. The drugs that are known to cause this sort of reaction are antibiotics such as penicillin, and nonsteroidal anti-inflammatory drugs, such as aspirin. The time between exposure to the drug and the development of acute tubulointerstitial nephritis can be anywhere from 5 days to 5 weeks. | ||
===Causes in Alphabetical Order=== | ===Causes in Alphabetical Order=== | ||
*[[ | {{columns-list|3| | ||
*[[Acetaminophen ]] | |||
*[[Acyclovir]] | *[[Acyclovir]] | ||
*[[Adenovirus]] | |||
*[[Aldomet]] | *[[Aldomet]] | ||
*[[Allopurinol]] | *[[Allopurinol]] | ||
*[[Alpha-interferon]] | *[[Alpha-interferon]] | ||
*[[Alport syndrome]] | *[[Alport syndrome]] | ||
*[[Amlodipine]] | *[[Amlodipine]] | ||
*[[Ampicillin]] | *[[Ampicillin]] | ||
*[[Anitrim]] | |||
*[[Anti-tubular basement membrane antibodies ]] | |||
*[[Apo-sulfatrim]] | |||
*[[Aristolochia]] | *[[Aristolochia]] | ||
*[[Aspirin]] | *[[Aspirin]] | ||
*[[Azathioprine]] | *[[Azathioprine]] | ||
*[[Bardet- | *[[Bactelan]] | ||
*[[Bardet-biedl syndrome]] | |||
*[[Batrizol]] | |||
*[[Brucella]] | |||
*[[Bumetanide]] | *[[Bumetanide]] | ||
*[[Candida]] | |||
*[[Captopril]] | *[[Captopril]] | ||
*[[Carbamazepine]] | *[[Carbamazepine]] | ||
*[[Cefaclor]] | *[[Cefaclor]] | ||
*[[ | *[[Cefotaxime sodium]] | ||
*[[ | *[[Cefoxitin sodium]] | ||
*[[Cephalosporin]] | |||
*[[Chlamydia]] | |||
*[[Chlorthalidone]] | *[[Chlorthalidone]] | ||
*[[ | *[[Cimetidine ]] | ||
*[[Ciprofloxacin]] | *[[Ciprofloxacin]] | ||
*[[Clofibrate]] | *[[Clofibrate]] | ||
*[[Cocaine]] | *[[Cocaine]] | ||
*[[ | *[[Coccidioidomycosis]] | ||
*[[Cytomegalovirus]] | *[[Corynebacterium diphtheriae]] | ||
*[[ | *[[Cox-2 inhibitors]] | ||
*[[Creatine]] | |||
*[[Cytomegalovirus]] | |||
*[[Dabrafenib mesylate]] | |||
*[[Diazepam]] | *[[Diazepam]] | ||
*[[Diclofenac]] | *[[Diclofenac]] | ||
*[[Diflunisal]] | *[[Diflunisal]] | ||
*[[Diltiazem]] | *[[Diltiazem]] | ||
*[[Diphenylhydantoin]] | *[[Diphenylhydantoin]] | ||
*[[Diphtheria]] | *[[Diphtheria]] | ||
*[[Diuretics]] | |||
*[[Doxycycline]] | *[[Doxycycline]] | ||
*[[Epstein- | *[[Ectaprim]] | ||
*[[Enterobacticel]] | |||
*[[Enterococcus]] | |||
*[[Epstein-barr virus ]] | |||
*[[Erythromycin]] | *[[Erythromycin]] | ||
*[[Escherichia coli]] | |||
*[[Esteprim]] | |||
*[[Ethambutol]] | *[[Ethambutol]] | ||
*[[Famotidine]] | *[[Famotidine]] | ||
*[[Fenoprofen]] | *[[Fenoprofen]] | ||
*[[Flurbiprofen]] | *[[Flurbiprofen]] | ||
*[[Francisella]] | |||
*[[Furosemide]] | *[[Furosemide]] | ||
*[[Granulomatosis with polyangiitis]] | |||
*[[Griseofulvin]] | *[[Griseofulvin]] | ||
*[[Hantaan virus]] | *[[Hantaan virus]] | ||
*[[ | *[[Histoplasmosis]] | ||
*[[Hiv infection]] | |||
*[[Hydralazine]] | *[[Hydralazine]] | ||
*[[Hydrochlorothiazide]] | *[[Hydrochlorothiazide]] | ||
*[[ | *[[Hypercalcemia]] | ||
* | *[[Hyperkalaemic distal renal tubular acidosis]] | ||
*[[Hyperuricemia]] | *[[Hyperuricemia]] | ||
*[[Hypokalemia]] | *[[Hypokalemia]] | ||
*[[Ibuprofen]] | *[[Ibuprofen]] | ||
*[[Igg4-related disease]] | |||
*[[Indinavir]] | *[[Indinavir]] | ||
*[[Indomethacin]] | *[[Indomethacin]] | ||
*[[Inflammatory bowel disease]] | |||
*[[Isobac]] | |||
*[[Isoniazid]] | *[[Isoniazid]] | ||
*[[Kawasaki's disease]] | *[[Kawasaki's disease]] | ||
*[[Kelfiprim]] | |||
*[[Ketoprofen]] | *[[Ketoprofen]] | ||
*[[Lansoprazole]] | *[[Lansoprazole]] | ||
| Line 68: | Line 94: | ||
*[[Macrolides]] | *[[Macrolides]] | ||
*[[Mesalamine]] | *[[Mesalamine]] | ||
*[[Mesalazine]] | *[[Mesalazine]] | ||
*[[Methicillin]] | *[[Methicillin]] | ||
*[[Metoxiprim]] | |||
*[[Minocycline hydrochloride]] | *[[Minocycline hydrochloride]] | ||
*[[ | *[[Mycobacterium tuberculosis]] | ||
*[[Mycoplasma infection]] | |||
*[[Myeloma]] | *[[Myeloma]] | ||
*[[Naproxen]] | *[[Naproxen]] | ||
*[[ | *[[Novo-trimel]] | ||
*[[Nu-cotrimox]] | |||
*[[Olsalazine]] | |||
*[[Omeprazole]] | *[[Omeprazole]] | ||
*[[Oxacillin]] | *[[Oxacillin]] | ||
*[[Oxaprozin]] | *[[Oxaprozin]] | ||
*[[Oxytetracycline]] | *[[Oxytetracycline]] | ||
*[[Pegylated interferon alfa-2b]] | *[[Pegylated interferon alfa-2b]] | ||
*[[Penicillin]] | *[[Penicillin ]] | ||
*[[Phenindione]] | *[[Phenindione]] | ||
*[[Phenobarbital]] | *[[Phenobarbital]] | ||
*[[Phenteramine]] | *[[Phenteramine]] | ||
*[[Phenylpropanolamine]] | *[[Phenylpropanolamine]] | ||
*[[Phenytoin]] | *[[Phenytoin]] | ||
*[[Piperacillin/tazobactam]] | |||
*[[Piroxicam]] | *[[Piroxicam]] | ||
*[[Polymyxin]] | *[[Polymyxin]] | ||
*[[ | *[[Polyomavirus]] | ||
*[[Propylthioruacil]] | *[[Pranlukast]] | ||
*[[Propylthioruacil]] | |||
*[[Pro-trin]] | |||
*[[Quinine]] | *[[Quinine]] | ||
*[[Rabeprazole]] | *[[Rabeprazole]] | ||
*[[Ranitidine]] | *[[Ranitidine]] | ||
*[[Reflux nephropathy]] | *[[Reflux nephropathy]] | ||
*[[Renal failure]] | *[[Renal failure]] | ||
*[[ | *[[Rickettsia infection]] | ||
*[[Rifampin]] | *[[Rifampin]] | ||
*[[ | *[[Roubac]] | ||
*[[Rubeola infection]] | |||
*[[Sarcoidosis]] | |||
*[[Sensenbrenner syndrome]] | *[[Sensenbrenner syndrome]] | ||
*[[Sicca syndrome]] | *[[Sicca syndrome]] | ||
| Line 106: | Line 139: | ||
*[[Streptococcal infection]] | *[[Streptococcal infection]] | ||
*[[Streptomycin]] | *[[Streptomycin]] | ||
*[[Sulfatrim ds]] | |||
*[[Sulfinpyrazone]] | *[[Sulfinpyrazone]] | ||
*[[Sulfonamide]] | |||
*[[Sulfoxaprim]] | |||
*[[Sulindac]] | *[[Sulindac]] | ||
*[[Syphilis]] | *[[Syphilis]] | ||
*[[Syraprim]] | |||
*[[Systemic lupus erythematosus]] | *[[Systemic lupus erythematosus]] | ||
* [[ | *[[Tenofovir disoproxil fumarate]] | ||
*[[Tetracycline]] | *[[Tetracycline]] | ||
*[[ | *[[Thiazide diuretics]] | ||
*[[Tolmetin]] | *[[Tolmetin]] | ||
*[[Toxoplasmosis]] | *[[Toxoplasmosis]] | ||
*[[Treponema]] | |||
*[[Triamterene]] | *[[Triamterene]] | ||
*[[ | *[[Trimesuxol]] | ||
*[[Trimethoprim-sulfamethoxazole]] | *[[Trimethoprim-sulfamethoxazole]] | ||
*[[ | *[[Trimetoger]] | ||
*[[Trimetox]] | |||
*[[Trimzol]] | |||
*[[Trisulfa]] | |||
*[[Trisulfam]] | |||
*[[Tubulointerstitial nephritis]] | |||
*[[Uroplus ds]] | |||
*[[Uroplus ss]] | |||
*[[Uveitis]] | |||
*[[Vancomycin]] | |||
*[[Wegener's granulomatosis]] | *[[Wegener's granulomatosis]] | ||
*[[Yersinia]] | |||
}} | |||
==References== | ==References== | ||
Revision as of 16:45, 23 November 2016
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Causes
Common causes include infection, or reaction to medication (such as an analgesic or antibiotics). 71%[1] to 92%[2] of cases are reported to be caused by drugs. This disease is also caused by other diseases and toxins that do damage to the kidney. Both acute and chronic tubulointerstitial nephritis can be caused by a bacterial infection in the kidneys, known as pyelonephritis. The most common cause is by an allergic reaction to a drug. The drugs that are known to cause this sort of reaction are antibiotics such as penicillin, and nonsteroidal anti-inflammatory drugs, such as aspirin. The time between exposure to the drug and the development of acute tubulointerstitial nephritis can be anywhere from 5 days to 5 weeks.
Causes in Alphabetical Order
The unnamed parameter 2= is no longer supported. Please see the documentation for {{columns-list}}.
3References
- ↑ Baker R, Pusey C (2004). "The changing profile of acute tubulointerstitial nephritis". Nephrol Dial Transplant. 19 (1): 8–11. PMID 14671029.
- ↑ Clarkson M, Giblin L, O'Connell F, O'Kelly P, Walshe J, Conlon P, O'Meara Y, Dormon A, Campbell E, Donohoe J (2004). "Acute interstitial nephritis: clinical features and response to corticosteroid therapy". Nephrol Dial Transplant. 19 (11): 2778–83. PMID 15340098.