Adrenolipoma pathophysiology: Difference between revisions
No edit summary |
No edit summary |
||
| Line 24: | Line 24: | ||
[[Category:Endocrine system]] | [[Category:Endocrine system]] | ||
[[Category:Endocrinology]] | [[Category:Endocrinology]] | ||
Revision as of 17:03, 9 September 2015
|
Adrenolipoma Microchapters |
|
Diagnosis |
|---|
|
Treatment |
|
Case Studies |
|
Adrenolipoma pathophysiology On the Web |
|
American Roentgen Ray Society Images of Adrenolipoma pathophysiology |
|
Risk calculators and risk factors for Adrenolipoma pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Ahmad Al Maradni, M.D. [2]
Overview
On gross pathology, central congested, red to brown lesion with thin cortical rim is a characteristic finding of adrenolipoma. On microscopic histopathological analysis, variable amounts of adipocytes and hematopietic cells are characteristic findings of adrenolipoma.
Pathogenesis
The most widely accepted theory in pathogenesis of adrenolipoma is the existence of metaplasia of the reticuloendothelial cells of blood capillaries in the adrenal gland in response to stimuli such as necrosis, infection, or stress.
Gross Pathology
Macroscopic examination demonstrates:[1]
Central congested, red to brown lesion with thin cortical rim, large lesions may contain haemorrhage or infarction
Microscopic Pathology
Histological examination demonstrates:[1]
- Variable amounts of mature adipocytes (with distended lipid vacuoles) similar to bone marrow
- Variable amounts of hematopoietic cells (including cells from myeloid, erythroid and megakaryocytic cells lines)