Polycystic ovary syndrome classification: Difference between revisions
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==Classification== | ==Classification== | ||
Two definitions are commonly used: | Two definitions are commonly used: | ||
*In 1990 a consensus workshop sponsored by the [[NIH]]/[[NICHD]] suggested that a patient has PCOS if she has | |||
**Signs of [[androgen]] excess (clinical or biochemical) | |||
**[[Oligoovulation]] | |||
**Other entities are excluded that would cause polycystic ovaries. | |||
*In 2003 a consensus workshop sponsored by [[ESHRE]]/[[ASRM]] in Rotterdam indicated PCOS to be present if 2 out of 3 criteria are met: <ref name="pmid14711538">{{cite journal |vauthors= |title=Revised 2003 consensus on diagnostic criteria and long-term health risks related to polycystic ovary syndrome |journal=Fertil. Steril. |volume=81 |issue=1 |pages=19–25 |year=2004 |pmid=14711538 |doi= |url=}}</ref> | |||
**[[Oligoovulation]] and/or [[anovulation]] | |||
**Excess androgen activity | |||
**Polycystic ovaries (by [[gynecologic ultrasound]]), and other causes of PCOS are excluded. | |||
The Rotterdam definition is wider, including many more patients, notably patients without androgen excess, whereas in the NIH/NICHD definition androgen excess is a prerequisite. Critics maintain that findings obtained from the study of patients with androgen excess cannot necessarily be extrapolated to patients without androgen excess. | The Rotterdam definition is wider, including many more patients, notably patients without androgen excess, whereas in the NIH/NICHD definition androgen excess is a prerequisite. Critics maintain that findings obtained from the study of patients with androgen excess cannot necessarily be extrapolated to patients without androgen excess. | ||
*PCOS is also classified based upon severity of symptoms into<ref name="pmid2893212">{{cite journal |vauthors=Jackson JR |title=Toxicity of herbicide containing glyphosate |journal=Lancet |volume=1 |issue=8582 |pages=414 |year=1988 |pmid=2893212 |doi= |url=}}</ref> | |||
**'''Asymptomatic form''': women with only PCO morphology | |||
**'''Mild form''': PCO morphology along with anovulation | |||
**'''Classical form''': Hyperandogenism along with ovarian dysfunction (anovulation and / or PCO) | |||
**'''Metabolic form''': Combination of mild and classical forms with presence of obesity and/or insulin resistance (abdominal obesity, insülin resistance, raised waist / hip ratio) | |||
==References== | ==References== | ||
Revision as of 19:20, 24 July 2017
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
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Overview
Classification
Two definitions are commonly used:
- In 1990 a consensus workshop sponsored by the NIH/NICHD suggested that a patient has PCOS if she has
- Signs of androgen excess (clinical or biochemical)
- Oligoovulation
- Other entities are excluded that would cause polycystic ovaries.
- In 2003 a consensus workshop sponsored by ESHRE/ASRM in Rotterdam indicated PCOS to be present if 2 out of 3 criteria are met: [1]
- Oligoovulation and/or anovulation
- Excess androgen activity
- Polycystic ovaries (by gynecologic ultrasound), and other causes of PCOS are excluded.
The Rotterdam definition is wider, including many more patients, notably patients without androgen excess, whereas in the NIH/NICHD definition androgen excess is a prerequisite. Critics maintain that findings obtained from the study of patients with androgen excess cannot necessarily be extrapolated to patients without androgen excess.
- PCOS is also classified based upon severity of symptoms into[2]
- Asymptomatic form: women with only PCO morphology
- Mild form: PCO morphology along with anovulation
- Classical form: Hyperandogenism along with ovarian dysfunction (anovulation and / or PCO)
- Metabolic form: Combination of mild and classical forms with presence of obesity and/or insulin resistance (abdominal obesity, insülin resistance, raised waist / hip ratio)