Aphthous ulcer pathophysiology: Difference between revisions
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It is thought that aphthous ulcer is the result of the cross reactivity between ''[[Staphylococcus sanguis]]'' and 60kDa Heat shock protein (HSP60) | It is thought that aphthous ulcer is the result of the cross reactivity between ''[[Staphylococcus sanguis]]'' and 60kDa Heat shock protein (HSP60) | ||
It is thought aphthous ulcer is a result of is caused by either , [hypothesis 2], or [hypothesis 3]. | |||
===Associated conditions=== | |||
Many diseases are associated with aphthous ulcer such as: | |||
==References== | ==References== |
Revision as of 13:01, 6 September 2016
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Sara Mehrsefat, M.D. [2]
Overview
The pathophysiology of Aphthous ulcers is not fully understood. It is though that reucurren
Pathophysiology
The exact pathogenesis of aphthous ulcer in not clear. Contributing factors include:
- Allergies
- Genetic predisposition
- Hormonal influences
- Hematologic abnormalities
- Immunologic factors
- Infectious agents
- Nutritional deficiencies
- Smoking cessation
- Stress
- Trauma
It is thought that aphthous ulcer is the result of the cross reactivity between Staphylococcus sanguis and 60kDa Heat shock protein (HSP60)
It is thought aphthous ulcer is a result of is caused by either , [hypothesis 2], or [hypothesis 3].
Associated conditions
Many diseases are associated with aphthous ulcer such as: