Esophageal candidiasis natural history, complications and prognosis: Difference between revisions
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==Complications== | ==Complications== | ||
Complications can be systemic due to spread of candida or localized due to the local invasion of the fungus. | |||
*Systemic spread and fungemia | *Systemic spread and fungemia | ||
*Esophageal perforation | *Esophageal perforation | ||
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==Prognosis== | ==Prognosis== | ||
*Prognosis is excellent | *Prognosis is excellent and Esophageal candidiaisis is usually responsive to antifungal treatment. | ||
*Recurrence is common after treatment due to persistence of the predisposing immunosuppression. | |||
==References== | ==References== |
Revision as of 14:10, 1 June 2017
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1];Associate Editor(s)-in-Chief: Ahmed Younes M.B.B.CH [2]
Overview
Candida esophagitis is very responsive to antifungal therapy. Complications can be systemic due to the spread of infection to the blood stream or local as esophageal stricture and obstruction.
Natural history
If left untreated, complications as esophageal stricture and candidaemia ensue.
Complications
Complications can be systemic due to spread of candida or localized due to the local invasion of the fungus.
- Systemic spread and fungemia
- Esophageal perforation
- Tracheoesophageal fistula
- Esophageal stricture
- Esophageal obstruction
Prognosis
- Prognosis is excellent and Esophageal candidiaisis is usually responsive to antifungal treatment.
- Recurrence is common after treatment due to persistence of the predisposing immunosuppression.