Esophageal candidiasis natural history, complications and prognosis: Difference between revisions
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==Complications== | ==Complications== | ||
Complications can be systemic due to spread of [[candida]] or localized due to the local invasion of the [[fungus]]. | Complications can be systemic due to spread of [[candida]] or localized due to the local invasion of the [[fungus]].<ref name="pmid3181663">{{cite journal |vauthors=Naito Y, Yoshikawa T, Oyamada H, Tainaka K, Morita Y, Kogawa T, Sugino S, Kondo M |title=Esophageal candidiasis |journal=Gastroenterol. Jpn. |volume=23 |issue=4 |pages=363–70 |year=1988 |pmid=3181663 |doi= |url=}}</ref> | ||
*Systemic spread and fungemia | *Systemic spread and fungemia | ||
*[[Esophageal perforation]] | *[[Esophageal perforation]] |
Latest revision as of 15:03, 1 June 2017
Esophageal candidiasis Microchapters |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1];Associate Editor(s)-in-Chief: Ahmed Younes M.B.B.CH [2]
Overview
Candida esophagitis is very responsive to antifungal therapy. Complications can be systemic due to the spread of infection to the blood stream or local as esophageal stricture and obstruction.
Natural history
If left untreated, complications as esophageal stricture and candidaemia ensue.
Complications
Complications can be systemic due to spread of candida or localized due to the local invasion of the fungus.[1]
- Systemic spread and fungemia
- Esophageal perforation
- Tracheoesophageal fistula
- Esophageal stricture
- Esophageal obstruction
Prognosis
- Prognosis is excellent and Esophageal candidiaisis is usually responsive to antifungal treatment.
- Recurrence is common after treatment due to persistence of the predisposing immunosuppression.