Diabetes insipidus historical perspective: Difference between revisions
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==Overview== | ==Overview== | ||
The history of | The history of diabetes insipidus dates as far back as the early 1670s when Thomas Willis noted that there was a difference in the taste of [[urine]] produced by different patients who presented with [[polyuria]] and [[polydipsia]] and this marked the beginning of the [[research]] into the difference between the popularly known [[diabetes mellitus]] and [[diabetes insipidus]]. | ||
==Historical Perspective== | ==Historical Perspective== | ||
*In | *In 1670, Thomas Willis, noted the difference in taste of [[urine]] from [[Polyuria|polyuric]] subjects compared with healthy individuals and started the differentiation of [[diabetes mellitus]] from [[Diabetes insipidus|diabetes insipidus.]] | ||
*In 1794, Johann Peter Frank described polyuric patients excreting nonsaccharine urine and introduced the term of | *In 1794, Johann Peter Frank described [[Polyuria|polyuric]] patients excreting nonsaccharine urine and introduced the term of diabetes insipidus. | ||
*In 1913, a historical milestone was achieved when Farini successfully used posterior pituitary extracts to treat | *In 1913, a historical milestone was achieved when Farini successfully used [[Posterior pituitary gland|posterior pituitary]] extracts to treat diabetesinsipidus. | ||
*In the early 1920s the available evidence indicated | *In the early 1920s the available evidence indicated diabetes insipidus as a disorder of the [[pituitary gland]]. | ||
*In the early 1928, De Lange first observed that some patients with | *In the early 1928, De Lange first observed that some patients with [[diabetes insipidus]] did not respond to [[posterior pituitary]] extracts. | ||
*Forssman and Waring in 1945 established that the kidney had a critical role for these forms of | *Forssman and Waring in 1945 established that the [[kidney]] had a critical role for these forms of diabetes insipidus resistant to this treatment. | ||
*In 1947 Williams and Henry introduced the term | *In 1947 Williams and Henry introduced the term [[nephrogenic diabetes insipidus]] for the [[Congenital syndromes|congenital syndrome]] characterized by [[polyuria]] and [[renal]] concentrating defect resistant to [[vasopressin]]. | ||
*In 1955, du Vigneaud received the 1955 Nobel Prize in chemistry for the first synthesis of the hormone vasopressin representing a milestone for the treatment of | *In 1955, du Vigneaud received the 1955 Nobel Prize in chemistry for the first synthesis of the hormone [[vasopressin]] representing a milestone for the treatment of [[central diabetes insipidus]].<ref name="pmid26913870">{{cite journal |vauthors=Valenti G, Tamma G |title=History of Diabetes Insipidus |journal=G Ital Nefrol |volume=33 Suppl 66 |issue= |pages=33.S66.1 |year=2016 |pmid=26913870 |doi= |url=}}</ref> | ||
==References== | ==References== | ||
Revision as of 12:25, 17 July 2017
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Overview
The history of diabetes insipidus dates as far back as the early 1670s when Thomas Willis noted that there was a difference in the taste of urine produced by different patients who presented with polyuria and polydipsia and this marked the beginning of the research into the difference between the popularly known diabetes mellitus and diabetes insipidus.
Historical Perspective
- In 1670, Thomas Willis, noted the difference in taste of urine from polyuric subjects compared with healthy individuals and started the differentiation of diabetes mellitus from diabetes insipidus.
- In 1794, Johann Peter Frank described polyuric patients excreting nonsaccharine urine and introduced the term of diabetes insipidus.
- In 1913, a historical milestone was achieved when Farini successfully used posterior pituitary extracts to treat diabetesinsipidus.
- In the early 1920s the available evidence indicated diabetes insipidus as a disorder of the pituitary gland.
- In the early 1928, De Lange first observed that some patients with diabetes insipidus did not respond to posterior pituitary extracts.
- Forssman and Waring in 1945 established that the kidney had a critical role for these forms of diabetes insipidus resistant to this treatment.
- In 1947 Williams and Henry introduced the term nephrogenic diabetes insipidus for the congenital syndrome characterized by polyuria and renal concentrating defect resistant to vasopressin.
- In 1955, du Vigneaud received the 1955 Nobel Prize in chemistry for the first synthesis of the hormone vasopressin representing a milestone for the treatment of central diabetes insipidus.[1]