Anxiety pathophysiology: Difference between revisions

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==Pathophysiology==
==Pathophysiology==
The cognitive component entails expectation of a diffuse and uncertain danger. Somatically the body prepares the organism to deal with threat (known as an emergency reaction): [[blood pressure]] and [[heart rate]] are increased, sweating is increased, bloodflow to the major muscle groups is increased, and [[immune system|immune]] and [[Digestion|digestive]] system functions are inhibited. Externally, somatic signs of anxiety may include pale skin, sweating, trembling, and[[mydriasis|pupillary dilation]]. Emotionally, anxiety causes a sense of dread or panic and physically causes nausea, and chills. Behaviorally, both voluntary and involuntary behaviors may arise directed at escaping or avoiding the source of anxiety and often maladaptive, being most extreme in [[anxiety disorder]]s. However, anxiety is not always pathological or maladaptive: it is a common emotion along with fear, anger, sadness, and happiness, and it has a very important function in relation to survival.
Neural circuitry involving the [[amygdala]] and [[hippocampus]] is thought to underlie anxiety.


==References==
==References==

Revision as of 14:45, 16 August 2017