Athlete's foot physical examination: Difference between revisions
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==== | ====Interdigital tinea pedis==== | ||
===== | *Most common type of athlete's foot is interdigital tinea pedis infection. | ||
*It is characterised by maceration, scaling, peeling of skin.<ref name="pmid26461834">{{cite journal| author=Canavan TN, Elewski BE| title=Identifying Signs of Tinea Pedis: A Key to Understanding Clinical Variables. | journal=J Drugs Dermatol | year= 2015 | volume= 14 | issue= 10 Suppl | pages= s42-7 | pmid=26461834 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=26461834 }} </ref> | |||
*Secondary bacterial infections are common. | |||
====Vesicolobullous tinea pedis==== | |||
*Bullae or tense vesicles are seen over the anterior plantar surface. | |||
*Pain is often an accompanying symptoms. | |||
*Vesicles appear in a symmetric fashion and may mimic pompholyx. | |||
*Dermatophytid reactions or identity reactions are most commonly seen in this type. | |||
*Secondary bacterial infections of ruptured bullae/vesicles may lead to ulcerative tinea pedis. | |||
====Chronic hyperkeratotic tinea pedis==== | |||
*Diffuse hyperkeratosis is the defining feature of chronic infection.<ref name="pmid2953766">{{cite journal| author=Greer DL, Gutierrez MM| title=Tinea pedis caused by Hendersonula toruloidea. A new problem in dermatology. | journal=J Am Acad Dermatol | year= 1987 | volume= 16 | issue= 5 Pt 2 | pages= 1111-5 | pmid=2953766 | doi=10.1016/s0190-9622(87)70144-3 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=2953766 }} </ref> | |||
*Skin is erythematous and has silvery scales. | |||
*Moccasin like distribution over the feet is seen, thus chronic intertriginous tinea is also called "moccasin" tinea pedis. | |||
====Ulcerative tinea pedis==== | |||
*This type is usually seen in diabetic patients. | |||
*It is characterised by ulcers, vesicles, pustules which begin in the interdigital web spaces which spread rapidly and may lead to a secondary bacterial infection. | |||
*Cellulitis, lymphangitis, edema are commonly seen with this type. | |||
<gallery> | <gallery> | ||
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Kiran Singh, M.D. [2]
Interdigital tinea pedis
- Most common type of athlete's foot is interdigital tinea pedis infection.
- It is characterised by maceration, scaling, peeling of skin.[1]
- Secondary bacterial infections are common.
Vesicolobullous tinea pedis
- Bullae or tense vesicles are seen over the anterior plantar surface.
- Pain is often an accompanying symptoms.
- Vesicles appear in a symmetric fashion and may mimic pompholyx.
- Dermatophytid reactions or identity reactions are most commonly seen in this type.
- Secondary bacterial infections of ruptured bullae/vesicles may lead to ulcerative tinea pedis.
Chronic hyperkeratotic tinea pedis
- Diffuse hyperkeratosis is the defining feature of chronic infection.[2]
- Skin is erythematous and has silvery scales.
- Moccasin like distribution over the feet is seen, thus chronic intertriginous tinea is also called "moccasin" tinea pedis.
Ulcerative tinea pedis
- This type is usually seen in diabetic patients.
- It is characterised by ulcers, vesicles, pustules which begin in the interdigital web spaces which spread rapidly and may lead to a secondary bacterial infection.
- Cellulitis, lymphangitis, edema are commonly seen with this type.
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
-
Tinea pedis. Adapted from Dermatology Atlas.[3]
References
- ↑ Canavan TN, Elewski BE (2015). "Identifying Signs of Tinea Pedis: A Key to Understanding Clinical Variables". J Drugs Dermatol. 14 (10 Suppl): s42–7. PMID 26461834.
- ↑ Greer DL, Gutierrez MM (1987). "Tinea pedis caused by Hendersonula toruloidea. A new problem in dermatology". J Am Acad Dermatol. 16 (5 Pt 2): 1111–5. doi:10.1016/s0190-9622(87)70144-3. PMID 2953766.
- ↑ 3.00 3.01 3.02 3.03 3.04 3.05 3.06 3.07 3.08 3.09 3.10 3.11 3.12 3.13 3.14 3.15 3.16 3.17 3.18 3.19 3.20 3.21 3.22 3.23 3.24 3.25 3.26 3.27 3.28 3.29 3.30 3.31 3.32 3.33 3.34 3.35 3.36 3.37 3.38 "Dermatology Atlas".