Diabetes insipidus historical perspective: Difference between revisions
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Revision as of 13:16, 27 September 2017
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Omodamola Aje B.Sc, M.D. [2]
Overview
The history of diabetes insipidus dates as far back as the early 1670s, when Thomas Willis noted that there was a difference in the taste of urine produced by different patients who presented with polyuria and polydipsia. This marked the beginning of the research into the difference between the popularly known diabetes mellitus and diabetes insipidus.
Historical Perspective
- In 1670, Thomas Willis, noted the difference in taste of urine from polyuric subjects compared with healthy individuals and began to differentiate diabetes mellitus from diabetes insipidus.
- In 1794, Johann Peter Frank described polyuric patients excreting nonsaccharine urine and introduced the term "diabetes insipidus."
- In 1913, a historical milestone was achieved when Farini successfully used posterior pituitary extracts to treat diabetes insipidus.
- In the early 1920s the available evidence indicated that diabetes insipidus was a disorder of the pituitary gland.
- In 1928, De Lange first observed that some patients with diabetes insipidus did not respond to posterior pituitary extracts.
- In 1945, Forssman and Waring established that the kidney played a critical role in the forms of diabetes insipidus that were resistant to this treatment.
- In 1947, Williams and Henry introduced the term nephrogenic diabetes insipidus for the congenital syndrome characterized by polyuria and renal concentrating defect resistant to vasopressin.
- In 1955, du Vigneaud received the 1955 Nobel Prize in chemistry for the first synthesis of the hormone vasopressin, which represented a milestone in the development of treatment for central diabetes insipidus.[1]