Sandbox: mahda20: Difference between revisions
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==Overview== | ==Overview<ref name="pmid5201062">{{cite journal |vauthors=Ahtaridis G, Snape WJ, Cohen S |title=Clinical and manometric findings in benign peptic strictures of the esophagus |journal=Dig. Dis. Sci. |volume=24 |issue=11 |pages=858–61 |year=1979 |pmid=520106 |doi= |url=}}</ref>== | ||
The exact pathogenesis of [disease name] is not fully understood. | The exact pathogenesis of [disease name] is not fully understood. | ||
OR | OR | ||
It is thought that | It is thought that esophageal stricture is the result of lower pressure of esophageal sphincter in [[gastroesophageal reflux disease]],esophageal motor disorder , or [hypothesis 3]. | ||
OR | OR | ||
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==Pathophysiology== | ==Pathophysiology== | ||
===Pathogenesis=== | ===Pathogenesis<ref name="pmid520106">{{cite journal |vauthors=Ahtaridis G, Snape WJ, Cohen S |title=Clinical and manometric findings in benign peptic strictures of the esophagus |journal=Dig. Dis. Sci. |volume=24 |issue=11 |pages=858–61 |year=1979 |pmid=520106 |doi= |url=}}</ref>=== | ||
*The exact pathogenesis of [disease name] is not fully understood. | *The exact pathogenesis of [disease name] is not fully understood. | ||
OR | OR | ||
*It is thought that | *It is thought that esophageal stricture is the result of lower pressure of esophageal sphincter in [[gastroesophageal reflux disease]], , esophageal motor disorderor, [hypothesis 3]. | ||
*[Pathogen name] is usually transmitted via the [transmission route] route to the human host. | *[Pathogen name] is usually transmitted via the [transmission route] route to the human host. | ||
*Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell. | *Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell. | ||
Revision as of 18:14, 18 October 2017
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Mahda Alihashemi M.D. [2]
Overview[1]
The exact pathogenesis of [disease name] is not fully understood.
OR
It is thought that esophageal stricture is the result of lower pressure of esophageal sphincter in gastroesophageal reflux disease,esophageal motor disorder , or [hypothesis 3].
OR
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
OR
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
OR
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
OR
The progression to [disease name] usually involves the [molecular pathway].
OR
The pathophysiology of [disease/malignancy] depends on the histological subtype.
Pathophysiology
Pathogenesis[2]
- The exact pathogenesis of [disease name] is not fully understood.
OR
- It is thought that esophageal stricture is the result of lower pressure of esophageal sphincter in gastroesophageal reflux disease, , esophageal motor disorderor, [hypothesis 3].
- [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
- Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
- [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
- The progression to [disease name] usually involves the [molecular pathway].
- The pathophysiology of [disease/malignancy] depends on the histological subtype.
Genetics
- [Disease name] is transmitted in [mode of genetic transmission] pattern.
- Genes involved in the pathogenesis of [disease name] include [gene1], [gene2], and [gene3].
- The development of [disease name] is the result of multiple genetic mutations.
Associated Conditions
Gross Pathology
- On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
Microscopic Pathology
- On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
References
- ↑ Ahtaridis G, Snape WJ, Cohen S (1979). "Clinical and manometric findings in benign peptic strictures of the esophagus". Dig. Dis. Sci. 24 (11): 858–61. PMID 520106.
- ↑ Ahtaridis G, Snape WJ, Cohen S (1979). "Clinical and manometric findings in benign peptic strictures of the esophagus". Dig. Dis. Sci. 24 (11): 858–61. PMID 520106.