Hepatocellular adenoma causes: Difference between revisions
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{{Hepatocellular adenoma}} | {{Hepatocellular adenoma}} | ||
{{CMG}}; {{AE}} {{ | {{CMG}}; {{AE}} {{ZAS}} | ||
==Overview== | ==Overview== | ||
==Causes== | ==Causes== | ||
* | * The causes of hepatocellular adenoma include; | ||
* | ** Oral contraceptive medications | ||
*** The causal relationship is proportional to the hormonal dose and duration of medication, highest in women over 30 years of age and after 24 months of using oral contraceptives. | |||
** Pregnancy | |||
*** It appears to stimulate rapid growth in these lesions with the risk of potentially fatal spontaneous rupture. | |||
** Glycogen storage disease types I,II and IV | |||
** Long term use of anabolic androgenic steroids | |||
** Metabolic syndrome | |||
** Maturity onset diabetes of young (MODY) | |||
** Obesity | |||
** Clomiphene | |||
** Familial adenomatous polyposis | |||
** Vascular disorders such as portal vein agenesis, budd chiari syndrome and hereditary hemorrhagic telangiectasia. | |||
==References== | ==References== | ||
{{reflist|1}} | {{reflist|1}} |
Revision as of 00:06, 28 December 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Zahir Ali Shaikh, MD[2]
Overview
Causes
- The causes of hepatocellular adenoma include;
- Oral contraceptive medications
- The causal relationship is proportional to the hormonal dose and duration of medication, highest in women over 30 years of age and after 24 months of using oral contraceptives.
- Pregnancy
- It appears to stimulate rapid growth in these lesions with the risk of potentially fatal spontaneous rupture.
- Glycogen storage disease types I,II and IV
- Long term use of anabolic androgenic steroids
- Metabolic syndrome
- Maturity onset diabetes of young (MODY)
- Obesity
- Clomiphene
- Familial adenomatous polyposis
- Vascular disorders such as portal vein agenesis, budd chiari syndrome and hereditary hemorrhagic telangiectasia.
- Oral contraceptive medications