Spontaneous coronary artery dissection risk factors: Difference between revisions
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* [[Connective tissue disorder]]: [[Marfan syndrome]], [[Loeys-Dietz syndrome]], [[Ehlers-Danlos syndrome|Ehlers-Danlos syndrome type 4]], [[neurofibromatosis type I]], [[cystic medial necrosis]], [[alpha-1 antitrypsin deficiency]], [[polycystic kidney disease]], [[lysyl oxidase|lysyl oxidase deficiency]], [[Alport syndrome]], [[pseudoxanthoma elasticum]] | * [[Connective tissue disorder]]: [[Marfan syndrome]], [[Loeys-Dietz syndrome]], [[Ehlers-Danlos syndrome|Ehlers-Danlos syndrome type 4]], [[neurofibromatosis type I]], [[cystic medial necrosis]], [[alpha-1 antitrypsin deficiency]], [[polycystic kidney disease]], [[lysyl oxidase|lysyl oxidase deficiency]], [[Alport syndrome]], [[pseudoxanthoma elasticum]] | ||
* [[Systemic inflammation|Systemic inflammatory disease]]: [[systemic lupus erythematosus]], [[Crohn's disease]], [[ulcerative colitis]], [[polyarteritis nodosa]], [[sarcoidosis]], [[Churg-Strauss syndrome]], [[Wegener's granulomatosis]], [[rheumatoid arthritis]], [[Kawasaki disease]], [[giant cell arteritis]], [[celiac disease]] | * [[Systemic inflammation|Systemic inflammatory disease]]: [[systemic lupus erythematosus]], [[Crohn's disease]], [[ulcerative colitis]], [[polyarteritis nodosa]], [[sarcoidosis]], [[Churg-Strauss syndrome]], [[Wegener's granulomatosis]], [[rheumatoid arthritis]], [[Kawasaki disease]], [[giant cell arteritis]], [[celiac disease]] | ||
* [[Hormonal therapy]]: [[oral contraceptive]], [[estrogen]], [[progesterone]], [[beta-HCG]], [[testosterone]], [[corticosteroids]] | * [[Hormonal therapy]]: [[oral contraceptive]], [[estrogen]], [[progesterone]], [[beta-HCG]], [[testosterone]], [[corticosteroids]], [[clomiphene]], [[polycystic ovarian syndrome]] | ||
* [[Takotsubo cardiomyopathy]] | * [[Takotsubo cardiomyopathy]] | ||
* [[Coronary artery spasm]] | * [[Coronary artery spasm]] | ||
Revision as of 01:10, 4 March 2018
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Spontaneous Coronary Artery Dissection Microchapters |
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Differentiating Spontaneous coronary artery dissection from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Type 1 Type 2A Type 2B Type 3 |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Nate Michalak, B.A. Arzu Kalayci, M.D. [2]
Synonyms and keywords: SCAD
Overview
The risk factors for spontaneous coronary artery dissection include predisposing factors (e.g., vasculopathy, pregnancy, connective tissue disorder, systemic inflammation) and precipitating stressors (e.g., strenuous exercise, emotional stress, recreational drugs).
Risk Factors
The phenotypic manifestation of spontaneous coronary artery dissection (SCAD) may occur as a result of predisposing factors compounded by precipitating stressors.[1] The presence of either predisposing or precipitating factors increases the risk of developing a dissection. The potential risk factors for SCAD include:[2]
References
- ↑ Saw, Jacqueline; Mancini, G.B. John; Humphries, Karin H. (2016). "Contemporary Review on Spontaneous Coronary Artery Dissection". Journal of the American College of Cardiology. 68 (3): 297–312. doi:10.1016/j.jacc.2016.05.034. ISSN 0735-1097.
- ↑ Saw, J.; Aymong, E.; Sedlak, T.; Buller, C. E.; Starovoytov, A.; Ricci, D.; Robinson, S.; Vuurmans, T.; Gao, M.; Humphries, K.; Mancini, G. B. J. (2014). "Spontaneous Coronary Artery Dissection: Association With Predisposing Arteriopathies and Precipitating Stressors and Cardiovascular Outcomes". Circulation: Cardiovascular Interventions. 7 (5): 645–655. doi:10.1161/CIRCINTERVENTIONS.114.001760. ISSN 1941-7640.