Bell's palsy pathophysiology: Difference between revisions
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==Genetics== | ==Genetics== | ||
*There is no established association between | *There is no established association between [[Genetics|genetic]] factors and Bell's palsy. | ||
==Associated Conditions== | ==Associated Conditions== | ||
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*On microscopic histopathological analysis, thickened [[perineurium]], infiltrates of inflammatory cells between nerve bundles and around [[blood vessel]]s are characteristic findings of Bell’s palsy. | *On microscopic histopathological analysis, thickened [[perineurium]], infiltrates of inflammatory cells between nerve bundles and around [[blood vessel]]s are characteristic findings of Bell’s palsy. | ||
* It appears that the histology of the [[facial nerve]] in Bell's palsy is similar to [[Herpes Zoster infection]], suggestive of an infectious cause. [[Category: (name of the system)]] | * It appears that the histology of the [[facial nerve]] in Bell's palsy is similar to [[Herpes Zoster infection]], suggestive of an infectious cause. [[Category: (name of the system)]] | ||
==References== | ==References== | ||
Revision as of 20:32, 26 April 2018
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Bell's palsy Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Bell's palsy pathophysiology On the Web |
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American Roentgen Ray Society Images of Bell's palsy pathophysiology |
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Risk calculators and risk factors for Bell's palsy pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Mohamadmostafa Jahansouz M.D.[2]
Overview
The exact pathogenesis of [disease name] is not fully understood.
OR
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
OR
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
OR
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
OR
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
OR
The progression to [disease name] usually involves the [molecular pathway].
OR
The pathophysiology of [disease/malignancy] depends on the histological subtype.
Pathophysiology
Pathogenesis
- It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
- [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
- Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
- [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
- The progression to [disease name] usually involves the [molecular pathway].
- The pathophysiology of [disease/malignancy] depends on the histological subtype.
Genetics
- There is no established association between genetic factors and Bell's palsy.
Associated Conditions
- Blepharospasm
- Blepharospasm has been rarely seen in patients whit with Bell's palsy.
- In most cases blepharospasm appeared within a month after the onset of Bell's palsy.
Gross Pathology
- On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
Microscopic Pathology
- On microscopic histopathological analysis, thickened perineurium, infiltrates of inflammatory cells between nerve bundles and around blood vessels are characteristic findings of Bell’s palsy.
- It appears that the histology of the facial nerve in Bell's palsy is similar to Herpes Zoster infection, suggestive of an infectious cause.