Renal tubular acidosis causes: Difference between revisions

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Revision as of 17:10, 14 May 2018

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Aditya Ganti M.B.B.S. [2]

	Primary Causes	Secondary Causes
Type 1	Idiopathic Familial Autosomal dominant Mainly due to mutations causing defects in the kidney anion exchanger [kAE1] in distal tubule intercalated cells. Autosomal recessive Mainly due to mutations causing defects in V-ATPase in distal tubule intercalated cells.	Autoimmune disorders Medications Ifosfamide Amphotericin B Lithium carbonate Ibuprofen Hypercalciuric conditions Hyperparathyroidism Vitamin D intoxication Sarcoidosis Idiopathic hypercalciuria Others

@@ Line 8: / Line 8: @@
 !Primary Causes
 !Secondary Causes
-!
 |-
 |Type 1
 |Idiopathic
 Familial
-* Autosomal dominant (mainly due to mutations causing defects in the kidney anion exchanger [kAE1] in distal tubule intercalated cells)
+* Autosomal dominant
-* Autosomal recessive (mainly due to mutations causing defects in V-ATPase in distal tubule intercalated cells)
+** Mainly due to mutations causing defects in the kidney anion exchanger [kAE1] in distal tubule intercalated cells.
-|
+* Autosomal recessive
+** Mainly due to mutations causing defects in V-ATPase in distal tubule intercalated cells.
 |
+* [[Autoimmune disorders]]
+* Medications
+** [[Ifosfamide]]
+** [[Amphotericin B]]
+** [[Lithium carbonate]]
+** [[Ibuprofen]]
+* Hypercalciuric conditions
+** [[Hyperparathyroidism]]
+** [[Vitamin D -- adverse effects|Vitamin D intoxication]]
+** [[Sarcoidosis]]
+** [[Idiopathic hypercalciuria]]
+* Others
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-|
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