Renal tubular acidosis causes: Difference between revisions

	Renal tubular acidosis Microchapters
Home
Patient Information
Overview
Historical Perspective
Classification
Pathophysiology
Causes
Differentiating Renal tubular acidosis from other Diseases
Epidemiology and Demographics
Risk Factors
Screening
Natural History, Complications and Prognosis
Diagnosis
Diagnostic Study of Choice
History and Symptoms
Physical Examination
Laboratory Findings
Electrocardiogram
X-ray
Echocardiography and Ultrasound
CT scan
MRI
Other Imaging Findings
Other Diagnostic Studies
Treatment
Medical Therapy
Surgery
Primary Prevention
Secondary Prevention
Cost-Effectiveness of Therapy
Future or Investigational Therapies
Case Studies
Case #1
Renal tubular acidosis causes On the Web
Most recent articles
Most cited articles
Review articles
CME Programs
Powerpoint slides
Images
American Roentgen Ray Society Images of Renal tubular acidosis causes All Images X-rays Echo & Ultrasound CT Images MRI
Ongoing Trials at Clinical Trials.gov
US National Guidelines Clearinghouse
NICE Guidance
FDA on Renal tubular acidosis causes
CDC on Renal tubular acidosis causes
Renal tubular acidosis causes in the news
Blogs on Renal tubular acidosis causes
Directions to Hospitals Treating Psoriasis
Risk calculators and risk factors for Renal tubular acidosis causes

Revision as of 17:30, 14 May 2018

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Aditya Ganti M.B.B.S. [2]

	Primary Causes	Secondary Causes
Type 1	Idiopathic Familial Autosomal dominant Mainly due to mutations causing defects in the kidney anion exchanger [kAE1] in distal tubule intercalated cells. Autosomal recessive Mainly due to mutations causing defects in V-ATPase in distal tubule intercalated cells.	Autoimmune disorders Medications Ifosfamide Amphotericin B Lithium carbonate Ibuprofen Hypercalciuric conditions Hyperparathyroidism Vitamin D intoxication Sarcoidosis Idiopathic hypercalciuria Others
Type 2	Recessive Proximal tubule cell sodium bicarbonate co-transporter (NBCe1) defect Carbonic anhydrase type 2 deficiency Cystinosis Tyrosinemia Hereditary fructose intolerance Galactosemia Glycogen storage disease (type I) Lowe syndrome	M-protein disorders Amyloidosis Multiple myeloma Medications Ifosfamide Tenofovir Acetazolamide Topiramate Aminoglycosides Heavy metals Lead Cadmium Mercury Copper Other Medullary sponge kidney Obstructive uropathy Renal transplant rejection Wilson disease
Type 4	Congenital hypoaldosteronism 21-hydroxylase deficiency Isolated hypoaldosteronism Pseudohypoaldosteronism type 2 Primary adrenal insufficiency Pseudohypoaldosteronism type 1	Angiotensin inhibitors, such as ACE inhibitors Angiotensin II receptor blockers Direct renin inhibitors Antibiotics, trimethoprim, and pentamidine Potassium-sparing diuretics Spironolactone Eplerenone Amiloride Triamterene

@@ Line 66: / Line 66: @@
 |Type 4
 |
-* Congenital hypoaldosteronism (21-hydroxylase deficiency and isolated hypoaldosteronism)
+* [[Congenital adrenal hyperplasia|Congenital hypoaldosteronism]]
-* Pseudohypoaldosteronism type 2 (Gordon's syndrome)
+** [[21-hydroxylase deficiency]]
-* Primary adrenal insufficiency
+** [[Isolated hypoaldosteronism]]
-* Pseudohypoaldosteronism type 1
+* [[Pseudohypoaldosteronism|Pseudohypoaldosteronism type 2]]
+* [[Primary adrenal insufficiency]]
+* [[Pseudohypoaldosteronism|Pseudohypoaldosteronism type 1]]
 |
-* Angiotensin inhibitors, such as ACE inhibitors, angiotensin II receptor blockers, and direct renin inhibitors
+* Angiotensin inhibitors, such as
-* Antibiotics, trimethoprim, and pentamidine
+** [[ACE inhibitor|ACE inhibitors]]
-* Potassium-sparing diuretics, such as spironolactone, eplerenone, amiloride, and triamterene
+** [[Angiotensin II receptor antagonist|Angiotensin II receptor blockers]]
+** Direct renin inhibitors
+* [[Antibiotics]], [[trimethoprim]], and [[pentamidine]]
+* Potassium-sparing diuretics
+** [[Spironolactone]]
+** [[Eplerenone]]
+** [[Amiloride]]
+** [[Triamterene]]
 |}
 ==References==
 {{reflist|2}}