Glucose-6-phosphate dehydrogenase deficiency risk factors: Difference between revisions
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==Risk Factors== | ==Risk Factors== | ||
Common risk factors in the development of G6PD deficiency include some foods such as fava beans, some medications and infections. | Common risk factors in the development of G6PD deficiency include some foods such as [[Fava bean|fava]] beans, some [[Medication|medications]] and [[Infection|infections]]. | ||
===Common Risk Factor=== | ===Common Risk Factor=== | ||
*Common risk factors in the development of G6PD include: | *Common risk factors in the development of G6PD include: |
Revision as of 17:13, 27 August 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Mahda Alihashemi M.D. [2]
Overview
There are no established risk factors for [disease name].
OR
The most potent risk factor in the development of [disease name] is [risk factor 1]. Other risk factors include [risk factor 2], [risk factor 3], and [risk factor 4].
OR
Common risk factors in the development of [disease name] include [risk factor 1], [risk factor 2], [risk factor 3], and [risk factor 4].
OR
Common risk factors in the development of [disease name] may be occupational, environmental, genetic, and viral.
Risk Factors
Common risk factors in the development of G6PD deficiency include some foods such as fava beans, some medications and infections.
Common Risk Factor
- Common risk factors in the development of G6PD include:
- Foods such as fava beans in G6PD mutation carriers
- Medications such as: [1]
- Aspirin
- Antimalarials: quinine, primaquine, pamaquine, and chloroquine
- Sulfonamides: mafenide, sulfanilamide, sulfamethoxazole
- Thiazolesulfone
- Methylene blue
- Analgesics: phenazopyridine and acetanilide
- Rasburicase
- Some non-sulfa antibiotics such as furazolidone, isoniazid, dapsone, nalidixic acid, nitrofurantoin).
- High dose vitamin C
- Bacterial, viral and rickettsial infection
- Some chemicals: Hanna in tattos and hair dyes.[2]
Less Common Risk Factors
- Less common risk factors in the development of G6PD include:
- Moth balls (naphthalene)
- Diabetic ketoacidosis[3]
- Amyl nitrite or isobutyl nitrite in RUSH ( sexual enhancement drug)[4]
References
- ↑ Richardson SR, O'Malley GF. PMID 29262208. Missing or empty
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(help) - ↑ Raupp P, Hassan JA, Varughese M, Kristiansson B (November 2001). "Henna causes life threatening haemolysis in glucose-6-phosphate dehydrogenase deficiency". Arch. Dis. Child. 85 (5): 411–2. PMC 1718961. PMID 11668106.
- ↑ Gellady AM, Greenwood RD (June 1972). "G-6-PD hemolytic anemia complicating diabetic ketoacidosis". J. Pediatr. 80 (6): 1037–8. PMID 4623682.
- ↑ Beaupre SR, Schiffman FJ (June 1994). "Rush hemolysis. A 'bite-cell' hemolytic anemia associated with volatile liquid nitrite use". Arch Fam Med. 3 (6): 545–8. PMID 8081534.
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [3]; Associate Editor(s)-In-Chief: Priyamvada Singh, M.D. [4]
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Overview
Risk Factors
- Ethnic groups
- African American
- Middle Eastern decent, particularly Kurdish or Sephardic Jewish
- Male
- Family history of the deficiency