Benign paroxysmal positional vertigo overview: Difference between revisions
Created page with "__NOTOC__ {{Benign paroxysmal positional vertigo}} {{CMG}}; {{AE}} {{Fs}} ==Overview== ==Historical Perspective== ==Classification== ==Pathophysiology== ==Causes== ==Diff..." |
No edit summary |
||
| Line 5: | Line 5: | ||
==Historical Perspective== | ==Historical Perspective== | ||
BPPV was first dicribed by Adler and Barany, who described it as a problem in the [[otolith]] [[organs]]. In 1952, Margaret Dix and Charles Hallpike named it [[positional nystagmus]] of the [[benign]] positional type. They noted [[nystagmus]] and [[vertigo]] with different [[head]]<nowiki/>movements. Hallpike also defined it as a peripheral problem rather than [[CNS|central]] ([[brain]]) problem. In 1962 Harold Schuknecht described theory of detached utricular [[otoconia]] (cupulolithiasis). Hall et al and Epley described the theory of free floating particle (canalithiasis). The first treatment strategy suggested for BPPV treatment was cawthorne's exercise (repeatitive [[head]] movement which cause [[vertigo]] in order to reach [[CNS|central]] adaption). The newest treatment strategy is to perform [[Dix-Hallpike test|Dix Hallpike test]] to diagnos and induce the vertigo and then performing CRP (Epley) maneuver. | |||
==Classification== | ==Classification== | ||
Benign paroxysmal positional vertigo may be classified according to which [[semicircular canal]] the [[otoconia]] have migrated to into 3 subtypes including [[posterior semicircular canal]] BPPV, [[lateral semicircular canal]] BPPV, and [[Superior semicircular canal|superior (anterior) semicircular canal]] BPPV. | |||
==Pathophysiology== | ==Pathophysiology== | ||
Revision as of 15:04, 6 March 2019
|
Benign paroxysmal positional vertigo Microchapters |
|
Differentiating Benign Paroxysmal Positional Vertigo from other Diseases |
|---|
|
Diagnosis |
|
Treatment |
|
Case Studies |
|
Benign paroxysmal positional vertigo overview On the Web |
|
American Roentgen Ray Society Images of Benign paroxysmal positional vertigo overview |
|
Risk calculators and risk factors for Benign paroxysmal positional vertigo overview |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Fahimeh Shojaei, M.D.
Overview
Historical Perspective
BPPV was first dicribed by Adler and Barany, who described it as a problem in the otolith organs. In 1952, Margaret Dix and Charles Hallpike named it positional nystagmus of the benign positional type. They noted nystagmus and vertigo with different headmovements. Hallpike also defined it as a peripheral problem rather than central (brain) problem. In 1962 Harold Schuknecht described theory of detached utricular otoconia (cupulolithiasis). Hall et al and Epley described the theory of free floating particle (canalithiasis). The first treatment strategy suggested for BPPV treatment was cawthorne's exercise (repeatitive head movement which cause vertigo in order to reach central adaption). The newest treatment strategy is to perform Dix Hallpike test to diagnos and induce the vertigo and then performing CRP (Epley) maneuver.
Classification
Benign paroxysmal positional vertigo may be classified according to which semicircular canal the otoconia have migrated to into 3 subtypes including posterior semicircular canal BPPV, lateral semicircular canal BPPV, and superior (anterior) semicircular canal BPPV.