Friedreich's ataxia risk factors: Difference between revisions
Jump to navigation
Jump to search
No edit summary |
No edit summary |
||
| Line 4: | Line 4: | ||
==Overview == | ==Overview == | ||
Because Friedreich’s Ataxia is a genetic diseases transmitted by autosomal recessive pattern, the most potent risk factor in the development of Friedreich’s Ataxia is strong family history. Other risk factors are unknown. The risk factors for developing associated clinical conditions of Friedreich's ataxia include: GAA1 length and age of diagnosis. | Because Friedreich’s Ataxia is a genetic diseases transmitted by [[autosomal recessive]] pattern, the most potent risk factor in the development of Friedreich’s Ataxia is strong family history. Other risk factors are unknown. The risk factors for developing associated clinical conditions of Friedreich's ataxia include: GAA1 length and age of diagnosis. | ||
==Risk Factors== | ==Risk Factors== | ||
* Because Friedreich’s Ataxia is a genetic diseases transmitted by autosomal recessive pattern, the most potent risk factor in the development of Friedreich’s Ataxia is strong family history. Other risk factors are unknown. | * Because Friedreich’s Ataxia is a genetic diseases transmitted by [[autosomal recessive]] pattern, the most potent risk factor in the development of Friedreich’s Ataxia is strong family history. Other risk factors are unknown. | ||
* The risk factors for developing associated clinical conditions of Friedreich's ataxia include:<ref name="pmid2579318">{{cite journal |vauthors=Lazo JS, Hait WN, Kennedy KA, Braun ID, Meandzija B |title=Enhanced bleomycin-induced DNA damage and cytotoxicity with calmodulin antagonists |journal=Mol. Pharmacol. |volume=27 |issue=3 |pages=387–93 |date=March 1985 |pmid=2579318 |doi= |url=}}</ref> | * The risk factors for developing associated clinical conditions of Friedreich's ataxia include:<ref name="pmid2579318">{{cite journal |vauthors=Lazo JS, Hait WN, Kennedy KA, Braun ID, Meandzija B |title=Enhanced bleomycin-induced DNA damage and cytotoxicity with calmodulin antagonists |journal=Mol. Pharmacol. |volume=27 |issue=3 |pages=387–93 |date=March 1985 |pmid=2579318 |doi= |url=}}</ref> | ||
** GAA1 length | ** GAA1 length | ||
Latest revision as of 20:16, 21 June 2019
|
Friedreich's ataxia Microchapters |
|
Diagnosis |
|---|
|
Treatment |
|
Case Studies |
|
Friedreich's ataxia risk factors On the Web |
|
American Roentgen Ray Society Images of Friedreich's ataxia risk factors |
|
Risk calculators and risk factors for Friedreich's ataxia risk factors |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Mohamadmostafa Jahansouz M.D
Overview
Because Friedreich’s Ataxia is a genetic diseases transmitted by autosomal recessive pattern, the most potent risk factor in the development of Friedreich’s Ataxia is strong family history. Other risk factors are unknown. The risk factors for developing associated clinical conditions of Friedreich's ataxia include: GAA1 length and age of diagnosis.
Risk Factors
- Because Friedreich’s Ataxia is a genetic diseases transmitted by autosomal recessive pattern, the most potent risk factor in the development of Friedreich’s Ataxia is strong family history. Other risk factors are unknown.
- The risk factors for developing associated clinical conditions of Friedreich's ataxia include:[1]
- GAA1 length
- Age of diagnosis