Dilated cardiomyopathy differential diagnosis

Dilated cardiomyopathy Microchapters
Home
Patient Information
Overview
Historical Perspective
Pathophysiology
Classification
Causes
Differentiating Dilated cardiomyopathy from other Diseases
Epidemiology and Demographics
Risk Factors
Screening
Natural History, Complications and Prognosis
Diagnosis
Diagnostic Study of Choice
History and Symptoms
Physical Examination
Laboratory Findings
Electrocardiogram
X-ray
Echocardiography and Ultrasound
CT scan
MRI
Other Imaging Findings
Other Diagnostic Studies
Treatment
Medical Therapy
Surgery
Primary Prevention
Secondary Prevention
Cost-Effectiveness of Therapy
Future or Investigational Therapies
Case Studies
Case #1
Dilated cardiomyopathy differential diagnosis On the Web
Most recent articles
Most cited articles
Review articles
CME Programs
Powerpoint slides
Images
American Roentgen Ray Society Images of Dilated cardiomyopathy differential diagnosis All Images X-rays Echo & Ultrasound CT Images MRI
Ongoing Trials at Clinical Trials.gov
US National Guidelines Clearinghouse
NICE Guidance
FDA on Dilated cardiomyopathy differential diagnosis
CDC on Dilated cardiomyopathy differential diagnosis
Dilated cardiomyopathy differential diagnosis in the news
Blogs on Dilated cardiomyopathy differential diagnosis
Directions to Hospitals Treating Dilated cardiomyopathy
Risk calculators and risk factors for Dilated cardiomyopathy differential diagnosis

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Abdelrahman Ibrahim Abushouk, MD[2]

Overview

Dilated cardiomyopathy should be differentiated from other causes of cardiac dysfunction

Differentiating Beriberi from other Diseases

Dilated cardiomyopathy should be differentiated from other causes of cardiac dysfunction

Disorders	Etiology	Clinical Presentation	Laboratory Findings
Acute Coronary Syndrome	Atherosclerosis Coronary thrombosis Plaque rupture	Chest Pain Dyspnea Palpitations Nausea, vomiting, and sweating	Echocardiogram: ST segment and T wave changes, pathological Q waves. Elevated blood troponin levels (after 6 hours of attack onset) Elevated blood CK-MB levels
Acute Pericarditis	Idiopathic Viral infection Connective tissue diseases (e.g. SLE) Dressler's Syndrome (after MI) Familial Mediterranean fever	Sharp chest pain (that increases with breathing/cough, reduced with leaning forward) Pericardial friction rub Fever (for inflammatory causes)	Echocardiogram: Slight ST segment elevation in several leads & diffuse T wave inversion CBC: Increased WBCs count Modest increase in CK-MB Elevated CRP levels.
Amphetamine/Cocaine Cardiomyopathy	Illicit drug use	History of drug use Chest pain Palpitation Other symptoms of amphetamine/cocaine intoxication	Drug and toxicology screen Elevated serum CK (rhabdomyolysis) Impaired electrolytes levels
Arrhythmogenic right ventricular cardiomyopathy (ARVC/D)	Idiopathic (but may be due to resolving myocarditis or genetic alterations in desmosomal proteins as plakoglobin and desmoplakin	Frequently symptomless until sudden cardiac death. Palpitations (Ventricular arrhythmia and conduction abnormalities) Exertional dyspnea Syncope	Diagnostic criteria are based on: Electrocardiogram Imaging modalities as 2D echocardiography and MRI.
Wet Beriberi	Inadequate thiamine intake (rice-based foods, alcoholism, and malnutrition) Increased thiamine loss (protracted vomiting) Inadequate absorption (after bariatric surgery or genetic mutation)	Palpitations Respiratory distress Edema of the legs Severe lactic acidosis.	Thiamine replacement test Reduced urinary thiamine Reduced erythrocyte transketolase and thiamine pyrophosphate activities.
Cardiac Tamponade	Ascending aortic dissection Collagen vascular diseases Iatrogenic - central line insertion, pacemaker insertion, coronary interventions, myocardial biopsy Idiopathic Malignancy - breast cancer, Kaposi's sarcoma, lung cancer, lymphomas Penetrating trauma Pericarditis After MI treatment (heparin and thrombolytics)	Acute tamponade: Cardiogenic shock, hypotension, cold extremities, peripheral cyanosis, and decreased urine output. Subacute tamponade: Peripheral edema with gradual progression to the aforementioned clinical picture.	Echocardiography to score the European Society of Cardiology (ESC) Working Group on Myocardial and Pericardial Diseases Increased serum CK-MB and troponin Cause-related investigations as serum inflammatory markers, diagnostic pericardiocentesis, and Gallium 67 imaging.
Thyrotoxicosis
Hypertrophic Cardiomyopathy
Left ventricular noncompaction
Myocarditis
Restrictive Cardiomyopathy