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{{familytree | |!| | | | |!| | | | | | |!| | | | | | | |!| | | | |}}
{{familytree | |!| | | | |!| | | | | | |!| | | | | | | |!| | | | |}}
{{familytree | G01 | | | G02 | | | | | G03 | | | | | | G04 | | | |G01=<div style="float: Center; text-align: Center; width: 28em; padding:1em;"> Classic triad of:
{{familytree | G01 | | | G02 | | | | | G03 | | | | | | G04 | | | |G01=<div style="float: Center; text-align: Center; width: 28em; padding:1em;"> Classic triad of:
:Hypotension <br> Elevated JVP <br> Clear Lungs <br>|
:Hypotension <br> Elevated JVP <br> Clear Lungs <br>
Kussmaul sign
Kussmaul sign
pulsus paradoxus
pulsus paradoxus

Revision as of 17:40, 5 August 2020

 
 
 
 
 
 
 
 
 
All patients with acute inferior wall myocardial infarction (ST elevation in leads II, III, aVF)
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Obtain right-sided precordial leads
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
>= 1mm ST elevation in lead V4R
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Highly suggestive of RVMI
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Physical examination
 
 
Echocardiography
 
 
 
 
Coronary Angiography
 
 
 
 
 
Hemodynamic study
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Classic triad of:
Hypotension
Elevated JVP
Clear Lungs

Kussmaul sign pulsus paradoxus Tricuspid regurgitation murmur Atrioventrcicular dissociation

Vagal symptoms: Bradycardia nausea Vomiting Diaphoresis

Pallor
 
 
Echocardiography
 
 
 
 
Coronary Angiography
 
 
 
 
 
Hemodynamic study