Blind loop syndrome pathophysiology: Difference between revisions
Jump to navigation
Jump to search
Pathophysiology |
|||
| Line 1: | Line 1: | ||
__NOTOC__ | __NOTOC__ | ||
==Overview== | ==Overview== | ||
<br /> | <br /> | ||
== Pathophysiology == | ==Pathophysiology== | ||
=== Physiology === | ===Physiology=== | ||
The normal physiology of [name of process] can be understood as follows: | The normal physiology of [name of process] can be understood as follows: | ||
=== Pathogenesis === | ===Pathogenesis=== | ||
* The exact pathogenesis of [disease name] is not completely understood. | *The exact pathogenesis of [disease name] is not completely understood. | ||
OR | OR | ||
* It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3]. | *It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3]. | ||
* [Pathogen name] is usually transmitted via the [transmission route] route to the human host. | *[Pathogen name] is usually transmitted via the [transmission route] route to the human host. | ||
* Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell. | *Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell. | ||
* [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells]. | *[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells]. | ||
* The progression to [disease name] | *The progression to [disease name] | ||
<br /> | <br /> | ||
== Associated Conditions == | ==Associated Conditions== | ||
Conditions associated with blind loop syndrome include: | Conditions associated with blind loop syndrome include: | ||
* Achlorhydria | *Achlorhydria | ||
*dysmotility | *dysmotility | ||
*fistulae | *fistulae | ||
*strictures | *strictures | ||
== Gross Pathology == | ==Gross Pathology== | ||
On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name]. | On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name]. | ||
== Microscopic Pathology == | ==Microscopic Pathology== | ||
On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name] | On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name] | ||
Revision as of 18:36, 29 August 2020
Overview
Pathophysiology
Physiology
The normal physiology of [name of process] can be understood as follows:
Pathogenesis
- The exact pathogenesis of [disease name] is not completely understood.
OR
- It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
- [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
- Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
- [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
- The progression to [disease name]
Associated Conditions
Conditions associated with blind loop syndrome include:
- Achlorhydria
- dysmotility
- fistulae
- strictures
Gross Pathology
On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
Microscopic Pathology
On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name]
Pathophysiology
The obstruction of a section of intestine causes ineffective bile salt mediated digestion of fats, causing fatty stools and poor absorption of fat and fat-soluble vitamins. Vitamin B12 deficiency may occur because the increased bacterial population can consume the vitamin.