Dyspareunia pathophysiology: Difference between revisions
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'''complex''': coital pain is greater than the simple peripheral tissue damage that may initially trigger the nociceptive component.When It becomes chronic,the pathophysiology of pain may gradually shift from nociceptive, a friend signal that should induce self protection and defense, to neuropathic, with a progressive involvement of the CNS. | '''complex''': coital pain is greater than the simple peripheral tissue damage that may initially trigger the nociceptive component.When It becomes chronic,the pathophysiology of pain may gradually shift from nociceptive, a friend signal that should induce self protection and defense, to neuropathic, with a progressive involvement of the CNS. | ||
==References== | ==References== | ||
Revision as of 20:52, 20 September 2020
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Dyspareunia Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Dyspareunia pathophysiology On the Web |
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American Roentgen Ray Society Images of Dyspareunia pathophysiology |
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Risk calculators and risk factors for Dyspareunia pathophysiology |
Overview
Dyspareunia is caused by medical causes, psychological problems, and pathophysiology can be considered as multifactorial, multisystemic or complex. dyspareunia vary based on the underlying causes . For example, estrogen deficiency causes atrophic vaginitis or ectopic uterine tissues in endometriosis causes dyspareunia.
Pathophysiology
Sexual pain disorders can therefore be considered as
- multifactorial
- multisystemic
- complex :
Multifactorial:biological, psychosexual, relational factors can coexist in a woman complaining of coital pain. Over time, these different factors may act as predisposing, precipitating, or perpetuating sexual pain disorders.
Multisystemic: sexual function involves:
- nervous,
- endocrine,
- vascular,
- immunological systems
- vaginal ecosystem
complex: coital pain is greater than the simple peripheral tissue damage that may initially trigger the nociceptive component.When It becomes chronic,the pathophysiology of pain may gradually shift from nociceptive, a friend signal that should induce self protection and defense, to neuropathic, with a progressive involvement of the CNS.